Morgellon's Syndrome 1st Edition
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Contents
Preface v
Introduction ix
1. Why the Investigation was Started 1
2. Materials and Methodology 5
3. Evidence not Recorded on Videotape 7
4. The Phenomenon of the Disappearing Hair Follicles 11
5. Particles Entering the Eyes 27
6. Particles Exiting the Eyes 41
7. A Substance that Transverses the Optic Globe 47
8. The Irregular Iris 57
9. Particles that Exit the Feet 59
10. Particles that Enter the Feet 75
11. The Microorganism Mobility–An Internal Propulsion System 87
12. Green Feet Illuminate the Role of the Epidermis in the Life-Cycle
of the Microorganism 95
13. Coated Hairs under the Microscope 97
14. The Micro Organism Possibly Growing on a Piece of Cocaine 105
15. A Summary of the Observations 107
16. The Structure of the Microorganism 113
17. A Model Accounting for the Observed Phenomena 115
18. Is There a Cure? 119
19. Future Considerations: A Microorganism as an Etiology for
Delusional Parasitosis 121
© 2010 by Taylor & Francis Group, LLC
Introduction
Every so often, scientific knowledge undergoes a revision. What was once thought
to be true is cast into doubt and sometimes new knowledge replaces the old. This
may be the eventual fate of delusional parasitosis.
Prior to 1994, peptic ulcers were thought to be caused by the hypersecretion of
gastric acid caused by increased but normal physiologic processes. The accepted
causes at the time of the hypersecretion of gastric acid included caffeine, spices,
alcohol, cigarette smoke and psychological stress. In 1982, Australian physicians
Robin Warren and Barry Marshall cultured Helicobacter pylori on a forgotten Petri
dish. To finally silence skeptics, Marshall drank a beaker of Helicobacter pylori to
demonstrate the role of this bacteria in the pathophysiology of gastritis and peptic
ulcers. Then in 1994, the National Institutes of Health (USA) recognized that most
cases of gastric ulcers were caused by Helicobacter pylori. Warren and Marshall
were awarded the Nobel Prize in Medicine in 2005 for their efforts.1
The high prevalence of Kaposi’s sarcoma, a form of dermatological cancer,
in AIDS patients pointed to a viral etiology, prompting research into a possible
causative agent. Then in 1994, Kaposi’s sarcoma was determined to be caused by
Human herpesvirus 8 (now known as Kaposi’s sarcoma-associated herpesvirus or
KSHV) by a team of Columbia University researchers.2
It is now time to reconsider the etiology of some or possibly all the cases of
delusional parasitosis. After a 15 month investigation, evidence of a microorganism
being the causative agent may have been discovered. Whether or not this
microorganism turns out to be the etiology of all cases of this disease, it is most
likely the cause of some of the cases, particularly in those cases involving cocaine
users.
Delusional parasitosis is defined as “a form of psychosis of false belief, a “loss
of contact with reality”. In delusional parasitosis, sufferers have a strong delusional
belief that they are infested with parasites, whereas in reality no such parasites are
present.”3 These patients present to dermatologists with specimens consisting of
1
http://en.wikipedia.org/wiki/Helicobacter_pylori
2
http://en.wikipedia.org/wiki/Kaposi’s_sarcoma
3
http://en.wikipedia.org/wiki/Delusional_parasitosis
© 2010 by Taylor & Francis Group, LLC
x Introduction
lint, skin or other debris contained in plastic saran wrap, celluloid tape, matchboxes
or other containers (the so-called “matchbox sign”).
Persons diagnosed with delusional parasitosis present with the belief that
parasites are crawling under their skin and sometimes exiting the dermis. Some
claim that materials such as hair and lint exit from their epidermis. The disease
is thought to be the result of psychotic delusions precipitated by mental illness
or stimulant drug use. In keeping with their perceived delusional origin, these
symptoms are currently treated with the antipsychotic medications risperidone
and olanzapine.4
The research reported in this book proves that inert objects such as hair, feathers
and lint and non-human white particles are entering the epidermis. Hairs, a feather
and white particles are also seen exiting the dermis and by extension, lint is most
likely also exiting the epidermis. The theories of how and why are discussed in the
analysis found in the final chapters of the book.
The investigation undertaken here examines the possibility that these symptoms
are not the result of delusions precipitated by the stimulant cocaine. Although the
case study presented here is a cocaine user, this may be incidental. It is unknown
whether cocaine is a prerequisite to be infected by the microorganism discussed
here and other persons diagnosed with delusional parasitosis have a separate
etiology, or whether cocaine only makes the host more susceptible than other
sufferers of delusional parasitosis to the same microorganism infecting non-users
of cocaine. Other possibilities include the possibility that cocaine users are more
aware of their symptoms or that the organism is more active in the presence of
cocaine and therefore more apparent and visible.
After global warming, cocaine is arguably the most deleterious substance on
the planet. In recent years, ten thousand Mexicans have died in violence attributed
to the drug trade. Colombians have been suffering from the terror accompanying
drug trafficking for decades. The human capacities of millions of Europeans,
Americans and Canadians have been diminished or totally eliminated by their
addiction to cocaine. It goes without saying that the world would be a much more
humane place if this scourge could be reduced or even eliminated.
I see the dehumanizing affects of cocaine addiction every day in my clinic
located in Parkdale, a drug affected neighborhood in Toronto. I see abused and
neglected children, usually brought in by social workers and foster parents, whose
parents are lost to their addictions. I am deeply saddened by formerly productive
human beings spending their whole lives in pursuit of the temporary euphoria
provided by cocaine. In the remote chance that an investigation into a possible
microorganism infecting cocaine users may help reduce cocaine use, I decided to
4
Noah S Scheinfeld, “Delusions of Parasitosis.” Available at http://emedicine.medscape.com/
article/1121818.
© 2010 by Taylor & Francis Group, LLC
Introduction xi
take the opportunity to investigate the strange complaints made by someone who
I suspected was consuming cocaine. Further inquiries confirmed that the patient
was in fact consuming cocaine.
With that background information, it was decided to investigate the complaints
made by a confirmed user of cocaine. The complaints consisted of the paradoxical
appearances of small patches of paroxysmal epidermal pruritis, the paroxysmal
paradoxical appearance of hair inside of socks and the sudden appearance of
abnormal domesticated feline behavior. These complaints were investigated in
both the human subject and his cat over a 16 month period using a video camera
and microscope.
Several totally unique phenomena were recorded including hair that appears to
be infected with what might turn out to be a fungal-type of microscopic organism.
The microorganism appears to have its own propulsion system and the ability to
enter and exit the epidermis, cornea and sclera rapidly. These specific phenomena
are present only in cocaine users and are elicited only in the presence of cocaine,
either after consumption by the host or as fumes in the surrounding atmosphere.
The distinct possibility exists that the subject has Morgellon’s disease5 which
is somehow amplified by the use of cocaine. Morgellon’s disease is the name
given to many cases of delusional parasitosis by people that believe that many
if not all cases of delusional parasitosis are not caused by delusions but have an
infectious etiology. The Centers for Disease Control and Prevention (CDC) began
an investigation into this unexplainable dermopathy. At the time of writing this
book, phase one of the study was completed and phase two of the investigation is
underway. Phase one consisted of subject evaluation and selection while phase two
consists of sample retrieval and analysis.
Important questions remain. How widespread is this organism in the
population? Is it responsible for, or contribute to, known or unknown diseases
such as Morgellon’s? Are there any medicinal or industrial uses for this organism?
And most importantly, can this organism consume cocaine and more specifically,
can it be utilized to eradicate cocaine plants? Further research is needed in order
to answer these and other questions that may arise.
5
“Mysterious ‘Morgellon’s’ disease prompts US investigation,” Emma Marris, Nature Medicine 12, 982
(1 September 2006).
© 2010 by Taylor & Francis Group, LLC
1
Why the Investigation was Started
This investigation was undertaken after several complaints of dermatological
abnormalities were made by cocaine users. The first complaint category was a
pruritic rash that was more pronounced while ingesting cocaine. The rash was
most pronounced on the extremities especially the distal aspects of the lower
limbs.
The second category of complaints was that they believed that mites were
shooting off the ceiling, landing on their heads and then burrowing into their
scalps. The complainant did not have to be actively ingesting cocaine to experience
this phenomenon, however, the complainant had to be in an area where cocaine
was chronically present. Non-cocaine users residing in the same household did
not experience the same symptoms. One complainant reported that her partner
had taken to sleeping in a tent set up in the living room in an effort to avoid this
symptom. Another complainant reported that she could hear the mites hitting the
floor as she walked by and that her cat was hiding under furniture to avoid them.
A third type of complaint was that mites were shooting off their cocaine pipes
while the cocaine was being vaporized and then burrowing into their faces. The
same phenomenon was described by most cocaine users not as burrowing mites,
but rather as impurities in bad cocaine. These complainants would occasionally
have several black spots on their faces after using the “bad” cocaine. This complaint
only occurred while using bad, crumbly cocaine bicarbonate and was not present
when using good quality, presumably uninfected, cocaine.
The last symptom shared by many complainants was that filamentous things
were coming out of their scalps while actively engaged in smoking cocaine. While
ingesting cocaine or shortly thereafter, the complainants reported that a small
patch of their scalps would begin to tingle. On occasion, within a couple of minutes
a large piece of lint would fall out of their hair. Similar symptoms also occurred
on their backs but without the expulsion of lint. Instead it felt like a large hair was
exiting their backs, sometimes lifting their shirts off their backs.
It should be stated here that two of the complainants were not cocaine users.
One was a grandmother concerned about a household infestation. The other was
© 2010 by Taylor & Francis Group, LLC
2 Morgellon's Syndrome
a middle-aged man experiencing the sensation of formication and white and
black materials exiting his skin. These would be non-stimulant using cases of
unexplained dermopathies, possibly Morgellon’s.
A telephone consultation with an infectious disease specialist, one of the world’s
leading experts on the controversial “cocaine mite,” was made in August 2007. He
reported that the dermatological symptoms experienced by cocaine smokers were
actually delusions brought on by the cerebral effects of cocaine which included
hallucinations and paranoia. He went on to say that the specimens he received from
the hair of his patients were, in reality, pieces of lint. The specialist reported that
the diagnosis was delusional parasitosis and the treatment was the administration
of the anti-psychotic and major tranquilizer medication risperidone at a dose of
1 mg by mouth daily. He went on to say that after the risperidone treatment was
initiated, the patients reported a reduction or cessation of their symptoms.
The effectiveness of risperidone in reducing the reported dermatological
symptoms by cocaine users may appear to confirm the etiology of these symptoms
as psychotic hallucinations. There are several alternative explanations for the
reduction in the reported symptomatology that occurs with risperidone therapy.
The first possible alternative reason is that risperidone is also known as a major
tranquilizer and the reduction in symptoms may be due to the tranquilizing effects
of the drug. The Compendium of Pharmaceuticals and Specialties lists somnolence
as a common side effect of risperidone.
Secondly, the symptoms may become less bothersome with time without
any treatment. The subject of this investigation reported a reduction in anxiety
associated with the symptomatology as time progressed. It appears that the
mechanism for the reduction in symptoms is similar to the mechanism of action
seen in desensitization therapy for anxiety disorders. The initial anxiety that
prompts the cocaine addict to seek medical attention seems to be rooted in the
belief that they are infested with an internal parasite that may be causing serious
damage to their health. After several months of experiencing the sensation of
focal paroxysmal paradoxical pruritis without adverse consequences, the research
subject stated that the anxiety and concern over the tingling subsided.
Thirdly, the effects of risperidone on symptom reduction may be psychological.
The majority of cocaine users consulted for this investigation believed that indeed
these symptoms were a result of the psychotic effects of cocaine. The reason for
this belief was most likely that consultations with infectious disease specialists
and dermatologists resulted in diagnoses of delusional parasitosis in those few
who sought treatment. Those cocaine addicts with the diagnosis then relate the
information to their colleagues and the theory of the symptoms arising from
hallucinations is widely accepted partly because they acknowledge that cocaine
causes other delusions as well as illusions and paranoia. The belief that the sensation
of parasitosis was a delusion caused by the cerebral effects of cocaine may be a sort
of reassurance to the infected individual.
© 2010 by Taylor & Francis Group, LLC
Why the Investigation was Started 3
At this point, a decision had to be made whether or not to investigate whether
the “cocaine mite” was in reality a result of psychotic hallucinatory delusions
brought on by the ingestion of cocaine or the result of an undiscovered parasite.
This was a very difficult decision to make. Cocaine is illegal and great risks were
involved in researching this substance without approval. In addition, the decision
to supply an individual an extremely dangerous and addictive substance was also
of questionable ethics.
The decision to commence an illegal research project was made for essentially
this reason: cocaine is responsible for the destruction of millions of lives worldwide.
The substance does not only affect the individual user but the whole of society.
Child abuse and neglect, murdered police officers and innocent bystanders, and
the loss of the user’s own humanity were all compelling reasons to investigate
these complaints. If this research and the consequent possibility of me becoming
publicly disgraced and possibly even incarcerated could save even one life it was
worth the effort. The cocaine trade is probably the root cause of most of the crime
and gang violence in the developed world. Colombia and Mexico have been
devastated by the violence associated with the production and distribution of the
drug. It was decided that the possibility of discovering an organism that could
be the beginning of the end for cocaine was important enough to commence an
investigation despite all the inherent risks.
With the ethical dilemma solved, or more accurately, rationalized, an anonymous
research subject was selected and supplied with between 600mg and 1000mg of
cocaine bicarbonate daily and the results were videotaped and specimens collected
and examined with a microscope.
The initial observation indicating that a newly discovered form of life exists
was seen in the research subject’s domestic feline. For no apparent reason, a feline
of the tuxedo type developed several previously unnoticed behaviors. First, the
subject feline began avoiding open areas of the habitation preferring to ambulate
throughout the house while remaining beneath furniture. Secondly, the subject cat
began exhibiting paroxysms of scratching and gnawing various parts of his body.
The third new behavior was that the cat developed paroxysmal tearing.
Subsequently, the human research subject noticed hair and white matter
emerging from his skin. Several specimens were sent to parasitology and no
organisms were detected. High definition videos of the subject reveal several
abnormal phenomena which most likely are caused by a unique previously
undiscovered microscopic organism.
© 2010 by Taylor & Francis Group, LLC
2
Materials and Methodology
To test the hypothesis that cocaine was responsible for this infection, cocaine was
incinerated on a metal copper pipe and the vaporized cocaine inhaled and then
blown onto the feet. The decision to expose the feet to the cocaine vapors was
made for two reasons. First of all, the tingling in the hair as well as the expulsion
of materials from the scalp occurred only while the subject was actively vaporizing
cocaine. The presence of cocaine seemed to stimulate whatever was causing these
phenomena. It was logical then to determine whether high concentrations of
cocaine molecules in the surrounding environment had any effect.
Secondly, the decision to expose the feet rather than, say, the arm was made
because the feet were the sites where hair was thought to be spontaneously
appearing inside of the socks. Initially, the hope was that the high concentrations
of cocaine vapors would induce the appearance of hair emerging from the soles
of the feet. However, despite expectations based on the above discussion, the
spontaneous emergence of hair from the feet was not witnessed throughout the
duration of the investigation.
The statement that no human hair was seen emerging from the feet during the
entire investigation is not entirely accurate. First of all, a strand of hair was seen
protruding from the plantar surface of the foot which then appeared to elongate
(see chapter 9). Secondly, hair was videotaped entering and then subsequently
exiting the epidermis (see chapter 4). Arguably, these two types of observations
could be classified as hair coming out of the epidermis of the feet. It is probably
better to say that no hair was observed exiting the epidermis of the feet prior
to the hair first being observed entering the epidermis of the feet and no hair
was observed exiting the feet prior to their appearance on the feet. This could
be explained by assuming that the hair only exits the feet while the feet are in
complete darkness. While no hair was seen exiting the feet, per se, lots of hair was
observed entering the epidermis of the feet. In addition, lots of other materials
were videotaped exiting and entering the feet (see chapters 4, 9 and 11).
The pedal phenomena were more pronounced if the feet were slightly moist and
were inside shoes just prior to the exposure to the vaporized cocaine. The videos
© 2010 by Taylor & Francis Group, LLC
6 Morgellon's Syndrome
of material spontaneously exiting the feet are all taken during or immediately
after exposure to the fumes of the incinerated cocaine. The enhancement of the
phenomenon in slightly moist feet was discovered by trial and error. The subject’s
feet were moist one day after stepping in puddle of water and there appeared to be
more particles exiting the feet while the feet remained moist.
To test the hypothesis that the microscopic organism was recruiting epithelial
cells in order to create some sort of extra-cellular matrix, green ink was applied to
the soles of the feet at one point during the investigation. The subject was examined
for changes in the dermis and eyes involving the color green.
Observations were recorded using a Sony HDD digital video recorder from
January 2008 until December 30, 2008. The videos were analyzed using the Sony
Picture Utility software supplied with the video camera. This software allowed the
frame by frame analyses needed to determine the mobility characteristics of this
rapidly moving micro organism. Consecutive Sony Picture Utility still frames are
separated by 33 milliseconds. A small number of videos are available on a DVD
included with the book along with the photographs contained within the book in
full color.
Large amounts of material extracted from the subject were collected in plastic
containers for future analysis.
© 2010 by Taylor & Francis Group, LLC
3
Evidence not Recorded on Videotape
The interviews with the complainants were elected to be placed in the introduction
however they could have just as easily been included with the observations. The
decision to place them in the introduction was made because most of them occurred
prior to the decision to commencing this investigation. The other problem was
that signed consents to reproduce and publish the informants’ complaints were
not obtained and therefore they must remain anonymous.
The observations were for the most part video taped, samples collected for
future analysis and slides of samples were made. However, some of the videotaped
data as well as all the collected samples and permanent slides made before April
2008 were deliberately discarded by the researcher’s spouse. Despite the lack of
physical evidence, it was decided to include the observations made by the research
subject and the descriptions of the discarded samples.
The research project commenced in late August 2007 when the research subject
began consuming between 600 mg and 1000 mg of vaporized cocaine daily
between the hours of 9pm and 3am. The cocaine was smoked in the basement
of his domicile where the subject also slept on a cot with a white cotton blanket.
About two to three weeks later, the subject noticed that minute white objects were
entering and exiting the ceiling halogen pot lights. At the time the white objects
were thought to be baby spiders and not related to the presence of cocaine. At the
time, the subject was expecting that the organism, if any did indeed exist, would
be in the form of a macroscopic parasite – the so-called “coca mite”.
About this time the subject began to experience paroxysms of paradoxical
pruritis especially immediately after vaporizing the cocaine. The subject’s feline
also began experiencing the same symptoms. Sometimes the subject’s cat would
sit down and begin gnawing various parts of his body especially just superior to
the dorsal base of his tail. A rash developed on the subject’s left ankle and when he
showed it to a co-worker she advised him that something appeared to be moving
through his lower leg soleus muscle.
An emergency physician was consulted and he suggested that the symptoms
were probably consistent with a subcutaneous vermis. He suggested taking vermox
© 2010 by Taylor & Francis Group, LLC
8 Morgellon's Syndrome
100 mg twice daily for 3 days and undergoing a course of permethrin 5% dermal
cream in case it was scabies. The advice was followed and vermox treatment started
the next day and the permethrin treatment the next evening. The next day nothing
abnormal was detected. The permethrin treatment and vermox were continued
that day. Again, on the third day nothing abnormal was detected. That day, the
vermox treatment was continued and instead of permethrin, a forty-five minute
soak from the neck down in a bathtub full of hot water with two cups of dissolved
Sunlight brand powdered laundry detergent was performed. Soaking in aqueous
Sunlight laundry detergent was thought to be an alternative treatment for scabies.
The next afternoon and over the next approximately 24 hours dozens of
C-shaped objects began to emerge from the dermis over the whole body from the
neck down. They were approximately 5 mm in total length but were only 2 mm
long because they were all folded in half resembling minute letters C. These objects
appeared to be composed of dozens of aligned white stiff fibers. These C-shaped
objects were saved using pieces of cellulose tape however they were all eventually
discarded by the subject’s spouse along with all the microscope slides and a jar of
samples.
The subject’s version of the origin of the C-shaped objects was met with
disbelief and attributed to hallucinations by the reviewers. It was at this point that
it was decided to purchase a video camera in order to record further phenomena
if they were to occur. Unfortunately, the phenomenon of the C-shaped objects
emerging from the dermis ended by the time a camera was procured and has not
reoccurred.
During the first six months of video-taping only one phenomenon was recorded
and retained (see video 37). All of the videotapes of the feline’s symptoms and all
but one video of the human subject were eventually erased when the 30 gigabyte
hard drive of the camera was full. After six months, an external hard drive was
purchased to store the rest of the videos and these videos make up the vast majority
of the videos described in this book.
The majority of the videos that were erased showed essentially one phenomenon:
specific longitudinal bands of epidermis located on the plantar surface of the
feet could be removed painlessly and then seemingly regenerate within seconds.
The painless character of the epidermal removal was not a consequence of the
normal inhalation of vaporized cocaine; rather it appears to be a direct result of
the presence of an inter-epidermal substance. The evidence that the lack of pain
wasn’t a result of the generalized analgesic effects of cocaine ingestion was that the
analgesia was localized. Indeed, the removal of the epidermis anywhere else except
in specific localized areas resulted in severe pain. Whether the analgesia was a
result of the intradermal keratin-microorganism matrix or the analgesic effects of
cocaine contained within the matrix or a combination of both is unknown.
The bands in the plantar surface of the feet began at the calcaneus, traveled
distally parallel to and within three centimeters of the lateral border of the foot
© 2010 by Taylor & Francis Group, LLC
Evidence not Recorded on Videotape 9
to about the proximal metatarsal heads. At this juncture, the band would move
medially to the area between the proximal heads of the first and second metatarsals
when the band would begin to move distal again for a couple of centimeters. At
this point between the band would move laterally again to about the middle of
the foot and would head distally again. The band was at this time located at the
proximal border of the metatarsal pad over the third metatarsal. From here, the
bands would move distally to the lower phalanges right to the toenails. At the
toenails the bands would travel proximally along the dorsal surface of the foot and
continue on towards the scalp.
The videos depicting the bands were sacrificed for two reasons. The first reason
was practical. In the early stages of the research there was nowhere to store the
data except in the hard drive of the camera. Admittedly, it was laziness, pure and
simple. I was working seven days a week, eleven hours a day, in my clinic seeing
patients and filming between four and six hours per day after work. The simple
act of reading the instructions provided with the camera, installing the software
to download the videos and purchasing more memory to store the videos was not
performed until more than three months into the video taping or seven months
into the research. Until then, less convincing videos were simply erased from the
camera’s thirty gigabyte hard drive when it became full in order to provide space
for more compelling evidence.
The second reason was an extension of the first reason. I was sacrificing the
less compelling videos out of laziness and it so happened that I decided that
videos depicting the removal of epidermal bands with a pair of needle forceps was
not compelling enough evidence. Indeed, I reviewed the films with a physician
colleague and he stated that all he could see was human mutilation.
Admittedly, the evidence of the bands in the videos was extremely subtle.
The evidence consisted of the removal of the band as it moved distally across the
metatarsal pads. The band would regenerate unless all the epidermis was removed.
If all the epidermis was removed then the band would simply move laterally to the
next strip of epidermis. The appearance of the regeneration of the band was subtle:
imagine slowly filling a collapsible garden hose with water. Basically, the thinned
out dermis became slightly thicker, not exactly compelling evidence of a parasitic
worm. I refer to a worm here because that was what I was expecting to find, a
worm in the dermis. After the initial samples were determined to be epithelium
(see figures 9.12 and 9.13), the methodology of epidermis removal was abandoned
in favor of focal material sampling and passive video observation.
The bands were not only located on the feet. The bands were also observed to
be located peri-orally extending between the oral labial angle and the nare along
both sides of both nasal-labial folds. These folds would become prominent while
ingesting cocaine - the left side more than the right - giving the face a distorted and
asymmetrical appearance previously attributed to involuntary muscle contractions
resulting from the neurological effects of cocaine ingestion.
© 2010 by Taylor & Francis Group, LLC
10 Morgellon's Syndrome
Fig. 3.1 This photograph depicts the typical facial distortion experienced by the subject
after ingesting cocaine. The fullness on the left side of the face was probably the result of
an intra-epidermal keratin-microorganism matrix that traveled through the subject. There is a
white particle on the epidermis located about 15 mm above the upper lip on the left side. The
left side of the upper lip appears swollen and the inferior aspect of the lip is white indicating
the possible presence of the microorganism.
The other category of video evidence that was not retained was the ocular
changes observed in a cat. The subject’s feline began to demonstrate paradoxical
and paradoxical ocular tearing combined with changes to the pupils and iris. The
cat was suspected of consuming cocaine crumbs on occasion. The camera was
placed by the cat’s bed and the eyes videotaped after vaporized cocaine was blown
into its face. White particles similar to the white particles seen in figure 6.7 were
seen exiting its eyes. The cat’s iris became irregular as though there was another
iris was located underneath his normal iris. The videos also showed that the cat’s
upper lip became noticeably swollen after ingesting cocaine mixed into its wet
food. This feature is analogous to the facial distortion seen in the human subject
(see figure 3.1). These videos were also sacrificed in the purges of the camera’s
memory.
These videos were deemed less compelling for two reasons. It was decided that
evidence from humans was more important and expertise in cat physiology was
not readily available. Again, the same physician colleague attributed the ocular
changes to feline peripheral cataracts after viewing the videos. At this point it was
decided to focus on collecting human evidence.
With the initial skepticism taken seriously, it was decided only to retain more
convincing evidence. Even though the evidence is referred to as more convincing,
it is acknowledged that there will always be credible criticism of the argument
made here that this constitutes evidence of a new micro organism. Therefore,
due to the anticipated controversial nature associated with any research of this
nature, hundreds of videos were compiled to corroborate the descriptions detailed
in this documentation of the investigation. Each video was assigned a number
by the camera and the selected videos will be referred to by these same numbers
preceded by. The photographs are taken from still frames of the videos produced
by the Sony Picture Utility software and color versions of these photographs are
included on the DVD accompanying the book. All of the videos described in the
book are included in the DVD that is provided with the book.
© 2010 by Taylor & Francis Group, LLC
4
The Phenomenon of the Disappearing
Hair Follicles
The paradoxical paroxysmal appearance of hair, mainly pubic hair, stuck on the
feet, was seen as an indication that something abnormal was occurring involving
the subject’s hair and also involving the feet. Although it was one of the first
observations, it was regarded as normal. This phenomenon was an observation
that took several months to recognize as abnormal because it is not abnormal to
see the occasional hair stuck to the bottom of a foot.
There were several abnormal characteristics regarding the phenomenon of the
hair on the feet that eventually lead to including it in the research. Until this time,
the hair was brushed off the feet with forceps and the subject’s hands after blowing
cocaine vapors onto the feet and prior to extracting material from the feet with the
forceps.
The hair was noted to be there immediately after removing the socks indicating
that the hair may or may not be entering the socks from within the feet. The
subject did not ambulate barefoot, donning socks immediately after bathing in the
morning, and therefore, there was little or no opportunity for hair to become stuck
to the feet. It should be stated here that this observation was made in retrospect
after it was determined that hair was indeed entering the dermis. By extension,
once hair was determined to be entering the subject through the epidermis, it
was surmised that the hair located on the feet was arriving by way of exiting the
dermis.
The observation that eventually led to the phenomenon being included in the
research was that there seemed to be much more pubic hair on the feet when
the pubic regions were pruritic. The connection between the appearance of
pubic hair located on the feet after relieving the pruritis with scratching and a
new phenomenon was made after a rather intense episode of pubic pruritis. After
that episode of pruritis, there were significantly more pubic hairs seen on the
plantar surface of the feet than usual. It was then decided to undergo a simple
experiment.
© 2010 by Taylor & Francis Group, LLC