TOPIC:ASCITES
Ascites
Accumulation of non-inflammatory fluid in the
peritoneal cavity characterized by bilateral
distension of lower abdomen and positive fluid
waves.
Aetiology
Primary causes:-
• Fall in osmotic pressure of blood
• Obstruction of lymphatic vessels
• Increased hydrostatic pressure in capillaries may be
due to cardiac insufficiency ,congestive heart failure
and passive congestion
Important aetiological factors
• Renal retention of sodium –
Renal insufficiency and liver damage.
• Hypoproteinaemia –
Protein deficiency diet
Lower protein synthesis in liver damage
Loss of protein in heavy parasitic infections like
haemonchosis , strongylosis.
Other important aetiological factors
Portal hypertension or lymphatic stasis
• In hepatic fibrosis, right side CHF, tricuspid valve
defects.
• Pressure on portal vein, posterior venacava ,
posterior vein and large abdominal lymph vessels
by abscesses, cyst, tumours etc.,
Pathogenesis
Venous stasis
Venous hypertension
Release of fluid
Accumulation of fluid in
peritoneal cavity
Hypoproteinaemia
Plasma colloidal osmotic pressure is decreased
Increase in hydrostatic pressure
Fluid escaped from circulation
Accumulated in body cavity
Liver Cirrhosis or Renal damage
Less kidney perfusion
Release of Rennin from kidney
Which act on angiotensin
Release of Aldosterone
Retention of more sodium in circulation
Ascites
Clinical findings
• Abdomen circumference -increased
• Bilateral distension of lower abdomen-
‘Pear shaped appearance’
• Linea alba-distended downward
• Flank region-hollowness with
prominent spines
• If animal body position is altered fluid
occupies most dependent part of
peritoneal cavity causing bulging at that
point
• Reduction in tone of abdominal muscles
• Dyspnoea, Constipation, inappetance
,concomittant anaemia, occasional
tympany
• Palpation-undulating movement of fluid
• Dogs and Cats:-
Distension may reach up to costal margins and
lower abdominal border may touch ground suface
Barrel shape-excessive distension
• Sheep and goat:-
Umbilicus - bulged outwards
Tactile percussion-fluid thrill or fluid waves
Subsequently animal shows
• Weakness , sunken eyes
• Increased respiration and heart rate
• Lie down as they have little tendency to
walk
• Engorged and prominently visible blood
vessels in abdominal area
• Death -Primary cause or cachectic
effects of generalised oedema
Necropsy findings
• Large amount of fluid in body cavity
• Adhesions in long standing cases
• Pressure on visceral organs also seen
Diagnosis
• History:- hypoproteinaemia
• Clinical signs:- liver cirrhosis
• Radiological examination:-
Hazy
Opaque abdominal cavity- ground glass
appearance
Abdominal organs may not be visible due to
presence of fluid having high specific gravity
• Ultrasonography :-
Ascitic fluid may be identified
• Abdominal paracentesis :-
Collect fluid by puncturing on midline by 14-15
gauze needle
Fluid-clear, watery, straw colored or turbid
containing fibrin, RBCs and WBCs.
Protein content below 3.5 g/dl and Specific gravity
less than 1.016
If milky- presence of lecithin
If turbid -presence of large no. Of cells
Greenish yellow-presence of bile due to as a result
of extravasation.
Reddish- presence of erythrocytes
Inflammatory-
If >500 total nucleated cells/ml of fluid in small
animals
If >9000cells/ml in large animals
Presence of macrophages
In haemorrhages- PCV15-20%
And Fluid clots due to presence of high prootein
Differential diagnosis
• Hepatomegaly
• Splenomegaly
• Uterine neoplasm
• Urethra obstruction with enlarged bladder -rectal
palpation
• Gastric torsion and intestinal obstruction
• Fat deposition
In al these cases abdominal enlargement is there
but fluid thrills are not felt and no shape alteration.
• Peritonitis- tenderness , high temp , paralytic ileus,
escape of exudate in abdominal paracentesis
Treatment
• In hypoproteinaemia associated with parasitic
infection
Broad spectrum antihelmintic
Protein rich diet-High biological value
Sodium free/low conc
• For fluid excretion
Diuretic therapy
Frusemide-0.5-2 ml small animals,
5-10ml in large animals,
I/M, once daily for 2-4
days
• Removal of excessive abdominal fluid-in
abdominal discomfort/ respiratory distress
puncture between umbilicus and pubis close to
linea alba by 14-15 gauze needle.
never drain completely- shock
0.5-2ml adrenaline-cardiac /respiratory failure
• Antibiotics
Ascites in domestic animals

Ascites in domestic animals

  • 1.
  • 2.
    Ascites Accumulation of non-inflammatoryfluid in the peritoneal cavity characterized by bilateral distension of lower abdomen and positive fluid waves.
  • 3.
    Aetiology Primary causes:- • Fallin osmotic pressure of blood • Obstruction of lymphatic vessels • Increased hydrostatic pressure in capillaries may be due to cardiac insufficiency ,congestive heart failure and passive congestion
  • 4.
    Important aetiological factors •Renal retention of sodium – Renal insufficiency and liver damage. • Hypoproteinaemia – Protein deficiency diet Lower protein synthesis in liver damage Loss of protein in heavy parasitic infections like haemonchosis , strongylosis.
  • 5.
    Other important aetiologicalfactors Portal hypertension or lymphatic stasis • In hepatic fibrosis, right side CHF, tricuspid valve defects. • Pressure on portal vein, posterior venacava , posterior vein and large abdominal lymph vessels by abscesses, cyst, tumours etc.,
  • 6.
    Pathogenesis Venous stasis Venous hypertension Releaseof fluid Accumulation of fluid in peritoneal cavity
  • 7.
    Hypoproteinaemia Plasma colloidal osmoticpressure is decreased Increase in hydrostatic pressure Fluid escaped from circulation Accumulated in body cavity
  • 8.
    Liver Cirrhosis orRenal damage Less kidney perfusion Release of Rennin from kidney Which act on angiotensin Release of Aldosterone Retention of more sodium in circulation Ascites
  • 9.
    Clinical findings • Abdomencircumference -increased • Bilateral distension of lower abdomen- ‘Pear shaped appearance’ • Linea alba-distended downward • Flank region-hollowness with prominent spines
  • 10.
    • If animalbody position is altered fluid occupies most dependent part of peritoneal cavity causing bulging at that point • Reduction in tone of abdominal muscles • Dyspnoea, Constipation, inappetance ,concomittant anaemia, occasional tympany • Palpation-undulating movement of fluid
  • 12.
    • Dogs andCats:- Distension may reach up to costal margins and lower abdominal border may touch ground suface Barrel shape-excessive distension • Sheep and goat:- Umbilicus - bulged outwards Tactile percussion-fluid thrill or fluid waves
  • 13.
    Subsequently animal shows •Weakness , sunken eyes • Increased respiration and heart rate • Lie down as they have little tendency to walk • Engorged and prominently visible blood vessels in abdominal area • Death -Primary cause or cachectic effects of generalised oedema
  • 14.
    Necropsy findings • Largeamount of fluid in body cavity • Adhesions in long standing cases • Pressure on visceral organs also seen
  • 15.
    Diagnosis • History:- hypoproteinaemia •Clinical signs:- liver cirrhosis • Radiological examination:- Hazy Opaque abdominal cavity- ground glass appearance Abdominal organs may not be visible due to presence of fluid having high specific gravity
  • 17.
  • 18.
    • Abdominal paracentesis:- Collect fluid by puncturing on midline by 14-15 gauze needle Fluid-clear, watery, straw colored or turbid containing fibrin, RBCs and WBCs. Protein content below 3.5 g/dl and Specific gravity less than 1.016
  • 19.
    If milky- presenceof lecithin If turbid -presence of large no. Of cells Greenish yellow-presence of bile due to as a result of extravasation. Reddish- presence of erythrocytes Inflammatory- If >500 total nucleated cells/ml of fluid in small animals If >9000cells/ml in large animals Presence of macrophages In haemorrhages- PCV15-20% And Fluid clots due to presence of high prootein
  • 20.
    Differential diagnosis • Hepatomegaly •Splenomegaly • Uterine neoplasm • Urethra obstruction with enlarged bladder -rectal palpation • Gastric torsion and intestinal obstruction • Fat deposition In al these cases abdominal enlargement is there but fluid thrills are not felt and no shape alteration. • Peritonitis- tenderness , high temp , paralytic ileus, escape of exudate in abdominal paracentesis
  • 21.
    Treatment • In hypoproteinaemiaassociated with parasitic infection Broad spectrum antihelmintic Protein rich diet-High biological value Sodium free/low conc • For fluid excretion Diuretic therapy Frusemide-0.5-2 ml small animals, 5-10ml in large animals, I/M, once daily for 2-4 days
  • 22.
    • Removal ofexcessive abdominal fluid-in abdominal discomfort/ respiratory distress puncture between umbilicus and pubis close to linea alba by 14-15 gauze needle. never drain completely- shock 0.5-2ml adrenaline-cardiac /respiratory failure • Antibiotics