CARDIOLOGY & EKGS
ARCHER NCLEX REVIEW
MUST KNOW SYSTEM FOR NCLEX SUCCESS!
ARCHER SLIDES ARE TO BE USED WITH RAPID FIRE/ CRASH COURSE TO
UNDERSTAND FULL CONCEPTS & APPLY IN NCLEX QUESTION SCENARIOS.
RAPID PREP WILL COVER THESE SLIDES AND EXPLAIN HIGHYIELD
CONCEPTS IN DETAIL
2 HOURS LIVE WEBINAR ON NOVEMBER 3RD, 2020
REGISTER AT WWW.ARCHERREVIEW.COM
ANATOMY & PHYSIOLOGY
BLOOD FLOW THROUGH THE HEART
HEMODYNAMICS
● Preload
○ Amount of blood returning to right side of the heart
● Afterload
○ Pressure against which the left ventricle must pump to eject blood
● Compliance
○ How easily the heart muscle expands when filled with blood
● Contractility
○ Strength of contraction of the heart muscle
● Stroke volume
○ Volume of blood pumped out of the ventricles with each contraction
● Cardiac output
○ The amount of blood the heart pumps through the circulatory system in a minute
CARDIAC OUTPUT
WHY is cardiac output SO important?!
● Tissue perfusion!
● End organ function
● Delivery of oxygen and nutrients to each and every cell in the body!
● Poor cardiac output??
○ Decreased LOC (not enough blood flow to the BRAIN)
○ Chest pain, weak peripheral pulses (not enough blood flow to the HEART)
○ SOB, crackles, rales (not enough blood flow to the LUNGS)
○ Cool, clammy, mottled extremities (not enough blood flow to the SKIN)
○ Decreased UOP (not enough blood flow to the KIDNEYS)
CO = SV X HR
CAUSES OF
DECREASED CO
● Increased blood volume...sometimes
● Tachycardia...sometimes
● Medications
○ ACE Inhibitors
○ ARBS
○ Nitrates
● Inotropes
CAUSES OF
INCREASED CO
● Bradycardia
● Arrhythmias
○ Pulseless v-tach
○ V-fib
○ Asystole
○ SVT
● Hypotension
● MI
● Cardiac muscle disease
EKG
ARCHER REVIEW CRASH COURSE
Normal Sinus Rhythm
P-wave: normal
PR Interval: 0.12-0.20
QRS: <0.12
Rate: 60-100
Regularity: Regular
Sinus Bradycardia
P-wave: Normal
PR Interval: 0.12-0.20
QRS: <0.12
Rate: <60
Regularity: Regular
Causes:
-Sleep
-Inactivity
-Very athletic
-Drugs
-MI
Sinus Tachycardia
P-wave: Normal
PR Interval: 0.12-0.20
QRS: <0.12
Rate: >100
Regularity: Regular
Causes:
-Caffeine
-Exercise
-Fever
-Anxiety
-Drugs
-Pain
-Hypotension
-Volume depletion
Atrial Flutter
P-wave: “saw-tooth”
PR Interval: none
QRS: <0.12
Rate: 250-400
Regularity: Regular or
Irregular
Causes:
-Heart disease
-MI
-CHF
-Pericarditis
Atrial Fibrillation
P-wave: ‘wavy’
PR Interval: none
QRS: <0.12
Rate: >400
Regularity: irregular
Causes:
-Heart Disease
-Pulmonary Disease
-Stress
-Alcohol
-Caffeine
Supraventricular Tachycardia (SVT)
P-wave: hidden
PR Interval:
immeasurable
QRS: <0.12
Rate: 150-250
Regularity: Regular
Causes:
-Caffeine
-CHF
-Fatigue
-Hypoxia
-Altered pacemaker in heart
Ventricular Tachycardia (V-Tach)
P-wave: none
PR Interval: none
QRS: >0.11 - ‘wide
complex & bizarre’
Rate: 150-250
Regularity: Regular
Causes:
-MI
-Ischemia
-Digoxin toxicity
-Hypoxia
-Acidosis
-Hypokalemia
-Hypotension
Ventricular Fibrillation (V-fib)
P-wave: none
PR Interval: none
QRS: none
Rate: none
Regularity: Irregular
Causes:
-MI
-Ischemia
-Hypoxia
-Acidosis
-Hypokalemia
-Hypotension
-Most common cause of sudden
death
Asystole
P-wave: possible to
have some random p-
waves
PR Interval: none
QRS: none
Rate: none
Regularity: n/a
Causes:
-Follows VT/VF in cardiac
arrest
-Acidosis
-Hypoxia
-Hypokalemia
-Hypothermia
-Overdose
CORONARY ARTERY DISEASE
(CAD)
WHAT IS CORONARY ARTERY DISEASE?
● The most common type of cardiovascular disease.
● Includes two types
○ Chronic stable angina
○ Acute coronary syndrome (aka MI)
CHRONIC STABLE ANGINA
● Chronic disease caused by narrowing of coronary
arteries and plaque build up.
● There are periods of decreased blood flow to the
heart muscle
● Decreased blood flow leads to decreased oxygen,
and ischemia.
● Ischemia causes chest pain
TREATMENT
● Nitroglycerin
○ Venous and arterial dilation → decreased afterload → increased CO
○ Given sublingual
○ Administer 1 pill q5 minutes for 3 doses
○ Do not swallow
○ Keep in a dark bottle in dry, cool place
○ Expected side effect = headache
EDUCATION
● DECREASE THE WORKLOAD OF THE HEART!
○ Rest
○ Do not overeat
○ No caffeine
○ Avoid temperature extremes
○ No smoking
○ Promote weight loss
○ Reduce stress
MYOCARDIAL INFARCTION
(MI)
WHAT IS A MYOCARDIAL INFARCTION
Myocardial infarction = acute coronary syndrome = unstable angina
● There is decreased blood flow to the heart, leading to decreased oxygen, and not only ischemia, but also
necrosis.
● Goal is to act quickly and limit the damage.
ASSESSMENT
● Chest pain
○ Crushing
○ Radiating to left arm or jaw
○ Between shoulder blades
● Epigastric discomfort/indigestion
● Fatigue
● SOB
● Vomiting
LABS
● CPK-MB
○ Cardiac specific isoenzyme
○ Increases with damage to cardiac cells
● Troponin
○ Cardiac biomarker
○ Increases with myocardial damage
● Myoglobin
○ Protein in muscle cells
○ Increases with damage to ANY muscle cell
○ Negative results can help rule out an MI, but positive results are not specific.
TREATMENT
● Cath lab within 90 minutes for PCI
○ Especially important if it’s a STEMI!
● Oxygen
● Aspirin
● Nitroglycerin
● Morphine
EDUCATION
● Quit smoking
● Increase activity gradually
● Diet
○ Low fat
○ Low salt
○ Low cholesterol
● Exercise
○ Avoid isometric exercises
○ Walking is a good choice
HEART FAILURE
WHAT IS HEART FAILURE?
The inability of the heart muscle to pump enough blood to meet the body's needs for blood and oxygen.
● Often results as a complication of other diseases
● #1 cause of HF is hypertension
● Other causes:
○ Cardiomyopathy
○ Endocarditis
○ MI
● Two types: Left and Right
LEFT-SIDED HEART FAILURE
Left side of the heart cannot move blood forward to the body.
Blood is backing up in the LUNGS.
Assessment:
● Pulmonary congestion
● Wet lung sounds
● Dyspnea
● Cough
● Blood tinged sputum
● S3
● Orthopnea
RIGHT HEART FAILURE
Right side of the heart cannot move blood forward to the lungs.
Blood is backing up in the BODY.
Assessment:
● Jugular venous distention
● Dependent edema
● Hepatomegaly
● Splenomegaly
● Ascites
● Weight gain
● Fatigue
● Anorexia
TREATMENT
● DECREASE THE WORKLOAD OF THE HEART!
● Primary strategy is to decrease afterload:
○ ACE Inhibitors
■ Arterial dilation→ decreased afterload → Increased stroke volume
○ ARBS
■ Decrease BP → decreased afterload → Increased CO
● Increase contractility
○ Digoxin
● Diuresis
○ Pt needs help reducing excess fluid
EDUCATION
● Take diuretic medications in the AM
● Monitor electrolyte levels while on diuretics
● Low sodium diet
○ This helps decrease fluid
● Elevate the HOB
○ Will help with diuresis
● Daily weight
○ Same time
○ Same scale
○ Same clothes
● Report any increase of 2-3 lbs in one day
HYPERTENSION
WHAT IS HYPERTENSION?
High blood pressure!
Normal <120/80
Elevated 120-129/80
Hypertension >130/>80
Hypertensive Crisis >180/>120
CAUSES & RISK FACTORS
● Family history
● African american race
● Increased age
● Obesity
● HLD
● CAD
● Stress
● Smoking
● High salt intake
● Caffeine
ASSESSMENT
● Often asymptomatic until very severe
● Vision changes
● Headaches
● Dizziness
● Nosebleeds
● SOB
● Angina
COMPLICATIONS
● Stroke
● MI
● Renal Failure
● Heart Failure
● Vision loss
TREATMENT & EDUCATION
● Medications
○ ACE inhibitors
○ Beta Blockers
○ CCB
○ Diuretics
● Diet
○ DASH
○ Low salt
○ Avoid caffeine and alcohol
○ Weight loss
○ Smoking cessation
● Lifestyle
○ Less sitting more walking
SHOCK
WHAT IS SHOCK??
● A state where the vital organs are not receiving adequate oxygenation.
● This lack of oxygenation causes organ damage and forces the cells to use anaerobic metabolism to create
energy….producing lactate.
● Cardiovascular system is composed of:
○ The blood
○ The vasculature
○ The heart
● A disruption in any of these three components can cause a lack of oxygen delivery to the organs, causing shock.
● Which component is ‘broken’ determines the type of shock.
TYPES OF SHOCK
Hypovolemic
Cardiogenic
Distributive
HYPOVOLEMIC SHOCK
PATHOPHYSIOLOGY
● Low blood flow
● There is a loss of the circulating volume
● Not enough blood to enter the heart (preload), which
decreases cardiac output.
● The body will vasoconstrict to compensate.
CAUSES
● Hemorrhage
● Traumatic injury
● Dehydration
○ Vomiting
○ Diarrhea
● Burns
ASSESSMENT
● Compensation
○ Weak
○ Pale
○ Tachycardic
○ Anxious
● Failing
○ Hypotension
○ Weak pulses
○ Tachycardic
○ Decreased LOC
○ Pale
○ Cool
○ Clammy
○ Decreased UOP
TREATMENT
● Fix the cause
○ Stop vomiting/diarrhea
○ Stop bleeding
■ Repair in OR
● Replace volume
○ Isotonic IVF
■ NS
■ LR
○ Blood products
● Support perfusion
○ Vasopressors
CARDIOGENIC SHOCK
PATHOPHYSIOLOGY
● The heart fails to pump sufficient blood out to the organs
● “Pump failure”
● Something is stopping the heart itself from getting blood out to the body
● Without sufficient blood delivered to the body, there is inadequate oxygenation
● Lack of oxygen impairs normal cellular metabolism
CAUSES
● MI
● Cardiac tamponade
● Pulmonary embolism
ASSESSMENT
● Decreased perfusion
○ Hypotension
○ Weak pulses
○ Cool, pale, clammy
○ Decreased UOP
○ Decreased LOC
● Volume overload
○ JVD
○ Crackles
○ SOB
○ Muffled heart sounds
○ S3
TREATMENT
● TREAT THE CAUSE
○ MI
■ PCI
■ CABG
○ PE
■ Thrombolytics
○ Tamponade
■ Pericardiocentesis
● Improve contractility
○ Dopamine
○ Dobutamine
● Decrease afterload
○ Diuretics
○ Dobutamine
● IABP
● LVAD
● Transplant
DISTRIBUTIVE SHOCK
PATHOPHYSIOLOGY
● Something causes an immune or autonomic response in the body
● This alters vascular tone
● The result is massive peripheral vasodilation
● With so much vasodilation, the blood pressure is inadequate to provide blood flow to the vital organs.
● Without sufficient blood delivered to the body, there is inadequate oxygenation
● Lack of oxygen impairs normal cellular metabolism
CAUSES
● Anaphylactic
○ Allergic reaction
● Neurogenic
○ SCI
● Septic
○ Systemic infection
○ Causes release of inflammatory cytokines
ASSESSMENT
● Decreased oxygen
● Hypotension
● Tachycardia
● Tachypnea
● Warm, flushed skin
● Decreased LOC
Specific:
● Anaphylactic
○ Hives
○ Rash
○ Swelling
○ Wheezing
● Neurogenic
○ SCI
○ Priapism
● Septic
○ Hyperthermic
○ Infection
TREATMENT
● Anaphylactic
○ Epinephrine
○ Corticosteroids
○ Bronchodilators
● Neurogenic
○ Cooling
○ Supportive care
● Septic
○ IV antibiotics
○ IVF

Cardiology and EKGs - Archer NCLEX crash course/ webinar

  • 1.
    CARDIOLOGY & EKGS ARCHERNCLEX REVIEW MUST KNOW SYSTEM FOR NCLEX SUCCESS! ARCHER SLIDES ARE TO BE USED WITH RAPID FIRE/ CRASH COURSE TO UNDERSTAND FULL CONCEPTS & APPLY IN NCLEX QUESTION SCENARIOS. RAPID PREP WILL COVER THESE SLIDES AND EXPLAIN HIGHYIELD CONCEPTS IN DETAIL 2 HOURS LIVE WEBINAR ON NOVEMBER 3RD, 2020 REGISTER AT WWW.ARCHERREVIEW.COM
  • 2.
  • 3.
  • 4.
    HEMODYNAMICS ● Preload ○ Amountof blood returning to right side of the heart ● Afterload ○ Pressure against which the left ventricle must pump to eject blood ● Compliance ○ How easily the heart muscle expands when filled with blood ● Contractility ○ Strength of contraction of the heart muscle ● Stroke volume ○ Volume of blood pumped out of the ventricles with each contraction ● Cardiac output ○ The amount of blood the heart pumps through the circulatory system in a minute
  • 5.
    CARDIAC OUTPUT WHY iscardiac output SO important?! ● Tissue perfusion! ● End organ function ● Delivery of oxygen and nutrients to each and every cell in the body! ● Poor cardiac output?? ○ Decreased LOC (not enough blood flow to the BRAIN) ○ Chest pain, weak peripheral pulses (not enough blood flow to the HEART) ○ SOB, crackles, rales (not enough blood flow to the LUNGS) ○ Cool, clammy, mottled extremities (not enough blood flow to the SKIN) ○ Decreased UOP (not enough blood flow to the KIDNEYS) CO = SV X HR
  • 6.
    CAUSES OF DECREASED CO ●Increased blood volume...sometimes ● Tachycardia...sometimes ● Medications ○ ACE Inhibitors ○ ARBS ○ Nitrates ● Inotropes CAUSES OF INCREASED CO ● Bradycardia ● Arrhythmias ○ Pulseless v-tach ○ V-fib ○ Asystole ○ SVT ● Hypotension ● MI ● Cardiac muscle disease
  • 7.
  • 8.
    Normal Sinus Rhythm P-wave:normal PR Interval: 0.12-0.20 QRS: <0.12 Rate: 60-100 Regularity: Regular
  • 9.
    Sinus Bradycardia P-wave: Normal PRInterval: 0.12-0.20 QRS: <0.12 Rate: <60 Regularity: Regular Causes: -Sleep -Inactivity -Very athletic -Drugs -MI
  • 10.
    Sinus Tachycardia P-wave: Normal PRInterval: 0.12-0.20 QRS: <0.12 Rate: >100 Regularity: Regular Causes: -Caffeine -Exercise -Fever -Anxiety -Drugs -Pain -Hypotension -Volume depletion
  • 11.
    Atrial Flutter P-wave: “saw-tooth” PRInterval: none QRS: <0.12 Rate: 250-400 Regularity: Regular or Irregular Causes: -Heart disease -MI -CHF -Pericarditis
  • 12.
    Atrial Fibrillation P-wave: ‘wavy’ PRInterval: none QRS: <0.12 Rate: >400 Regularity: irregular Causes: -Heart Disease -Pulmonary Disease -Stress -Alcohol -Caffeine
  • 13.
    Supraventricular Tachycardia (SVT) P-wave:hidden PR Interval: immeasurable QRS: <0.12 Rate: 150-250 Regularity: Regular Causes: -Caffeine -CHF -Fatigue -Hypoxia -Altered pacemaker in heart
  • 14.
    Ventricular Tachycardia (V-Tach) P-wave:none PR Interval: none QRS: >0.11 - ‘wide complex & bizarre’ Rate: 150-250 Regularity: Regular Causes: -MI -Ischemia -Digoxin toxicity -Hypoxia -Acidosis -Hypokalemia -Hypotension
  • 15.
    Ventricular Fibrillation (V-fib) P-wave:none PR Interval: none QRS: none Rate: none Regularity: Irregular Causes: -MI -Ischemia -Hypoxia -Acidosis -Hypokalemia -Hypotension -Most common cause of sudden death
  • 16.
    Asystole P-wave: possible to havesome random p- waves PR Interval: none QRS: none Rate: none Regularity: n/a Causes: -Follows VT/VF in cardiac arrest -Acidosis -Hypoxia -Hypokalemia -Hypothermia -Overdose
  • 17.
  • 18.
    WHAT IS CORONARYARTERY DISEASE? ● The most common type of cardiovascular disease. ● Includes two types ○ Chronic stable angina ○ Acute coronary syndrome (aka MI)
  • 19.
    CHRONIC STABLE ANGINA ●Chronic disease caused by narrowing of coronary arteries and plaque build up. ● There are periods of decreased blood flow to the heart muscle ● Decreased blood flow leads to decreased oxygen, and ischemia. ● Ischemia causes chest pain
  • 20.
    TREATMENT ● Nitroglycerin ○ Venousand arterial dilation → decreased afterload → increased CO ○ Given sublingual ○ Administer 1 pill q5 minutes for 3 doses ○ Do not swallow ○ Keep in a dark bottle in dry, cool place ○ Expected side effect = headache
  • 21.
    EDUCATION ● DECREASE THEWORKLOAD OF THE HEART! ○ Rest ○ Do not overeat ○ No caffeine ○ Avoid temperature extremes ○ No smoking ○ Promote weight loss ○ Reduce stress
  • 22.
  • 23.
    WHAT IS AMYOCARDIAL INFARCTION Myocardial infarction = acute coronary syndrome = unstable angina ● There is decreased blood flow to the heart, leading to decreased oxygen, and not only ischemia, but also necrosis. ● Goal is to act quickly and limit the damage.
  • 24.
    ASSESSMENT ● Chest pain ○Crushing ○ Radiating to left arm or jaw ○ Between shoulder blades ● Epigastric discomfort/indigestion ● Fatigue ● SOB ● Vomiting
  • 25.
    LABS ● CPK-MB ○ Cardiacspecific isoenzyme ○ Increases with damage to cardiac cells ● Troponin ○ Cardiac biomarker ○ Increases with myocardial damage ● Myoglobin ○ Protein in muscle cells ○ Increases with damage to ANY muscle cell ○ Negative results can help rule out an MI, but positive results are not specific.
  • 26.
    TREATMENT ● Cath labwithin 90 minutes for PCI ○ Especially important if it’s a STEMI! ● Oxygen ● Aspirin ● Nitroglycerin ● Morphine
  • 27.
    EDUCATION ● Quit smoking ●Increase activity gradually ● Diet ○ Low fat ○ Low salt ○ Low cholesterol ● Exercise ○ Avoid isometric exercises ○ Walking is a good choice
  • 28.
  • 29.
    WHAT IS HEARTFAILURE? The inability of the heart muscle to pump enough blood to meet the body's needs for blood and oxygen. ● Often results as a complication of other diseases ● #1 cause of HF is hypertension ● Other causes: ○ Cardiomyopathy ○ Endocarditis ○ MI ● Two types: Left and Right
  • 30.
    LEFT-SIDED HEART FAILURE Leftside of the heart cannot move blood forward to the body. Blood is backing up in the LUNGS. Assessment: ● Pulmonary congestion ● Wet lung sounds ● Dyspnea ● Cough ● Blood tinged sputum ● S3 ● Orthopnea
  • 31.
    RIGHT HEART FAILURE Rightside of the heart cannot move blood forward to the lungs. Blood is backing up in the BODY. Assessment: ● Jugular venous distention ● Dependent edema ● Hepatomegaly ● Splenomegaly ● Ascites ● Weight gain ● Fatigue ● Anorexia
  • 32.
    TREATMENT ● DECREASE THEWORKLOAD OF THE HEART! ● Primary strategy is to decrease afterload: ○ ACE Inhibitors ■ Arterial dilation→ decreased afterload → Increased stroke volume ○ ARBS ■ Decrease BP → decreased afterload → Increased CO ● Increase contractility ○ Digoxin ● Diuresis ○ Pt needs help reducing excess fluid
  • 33.
    EDUCATION ● Take diureticmedications in the AM ● Monitor electrolyte levels while on diuretics ● Low sodium diet ○ This helps decrease fluid ● Elevate the HOB ○ Will help with diuresis ● Daily weight ○ Same time ○ Same scale ○ Same clothes ● Report any increase of 2-3 lbs in one day
  • 34.
  • 35.
    WHAT IS HYPERTENSION? Highblood pressure! Normal <120/80 Elevated 120-129/80 Hypertension >130/>80 Hypertensive Crisis >180/>120
  • 36.
    CAUSES & RISKFACTORS ● Family history ● African american race ● Increased age ● Obesity ● HLD ● CAD ● Stress ● Smoking ● High salt intake ● Caffeine
  • 37.
    ASSESSMENT ● Often asymptomaticuntil very severe ● Vision changes ● Headaches ● Dizziness ● Nosebleeds ● SOB ● Angina
  • 38.
    COMPLICATIONS ● Stroke ● MI ●Renal Failure ● Heart Failure ● Vision loss
  • 39.
    TREATMENT & EDUCATION ●Medications ○ ACE inhibitors ○ Beta Blockers ○ CCB ○ Diuretics ● Diet ○ DASH ○ Low salt ○ Avoid caffeine and alcohol ○ Weight loss ○ Smoking cessation ● Lifestyle ○ Less sitting more walking
  • 41.
  • 42.
    WHAT IS SHOCK?? ●A state where the vital organs are not receiving adequate oxygenation. ● This lack of oxygenation causes organ damage and forces the cells to use anaerobic metabolism to create energy….producing lactate. ● Cardiovascular system is composed of: ○ The blood ○ The vasculature ○ The heart ● A disruption in any of these three components can cause a lack of oxygen delivery to the organs, causing shock. ● Which component is ‘broken’ determines the type of shock.
  • 43.
  • 45.
  • 46.
    PATHOPHYSIOLOGY ● Low bloodflow ● There is a loss of the circulating volume ● Not enough blood to enter the heart (preload), which decreases cardiac output. ● The body will vasoconstrict to compensate.
  • 47.
    CAUSES ● Hemorrhage ● Traumaticinjury ● Dehydration ○ Vomiting ○ Diarrhea ● Burns
  • 48.
    ASSESSMENT ● Compensation ○ Weak ○Pale ○ Tachycardic ○ Anxious ● Failing ○ Hypotension ○ Weak pulses ○ Tachycardic ○ Decreased LOC ○ Pale ○ Cool ○ Clammy ○ Decreased UOP
  • 49.
    TREATMENT ● Fix thecause ○ Stop vomiting/diarrhea ○ Stop bleeding ■ Repair in OR ● Replace volume ○ Isotonic IVF ■ NS ■ LR ○ Blood products ● Support perfusion ○ Vasopressors
  • 50.
  • 51.
    PATHOPHYSIOLOGY ● The heartfails to pump sufficient blood out to the organs ● “Pump failure” ● Something is stopping the heart itself from getting blood out to the body ● Without sufficient blood delivered to the body, there is inadequate oxygenation ● Lack of oxygen impairs normal cellular metabolism
  • 52.
    CAUSES ● MI ● Cardiactamponade ● Pulmonary embolism
  • 53.
    ASSESSMENT ● Decreased perfusion ○Hypotension ○ Weak pulses ○ Cool, pale, clammy ○ Decreased UOP ○ Decreased LOC ● Volume overload ○ JVD ○ Crackles ○ SOB ○ Muffled heart sounds ○ S3
  • 54.
    TREATMENT ● TREAT THECAUSE ○ MI ■ PCI ■ CABG ○ PE ■ Thrombolytics ○ Tamponade ■ Pericardiocentesis ● Improve contractility ○ Dopamine ○ Dobutamine ● Decrease afterload ○ Diuretics ○ Dobutamine ● IABP ● LVAD ● Transplant
  • 55.
  • 56.
    PATHOPHYSIOLOGY ● Something causesan immune or autonomic response in the body ● This alters vascular tone ● The result is massive peripheral vasodilation ● With so much vasodilation, the blood pressure is inadequate to provide blood flow to the vital organs. ● Without sufficient blood delivered to the body, there is inadequate oxygenation ● Lack of oxygen impairs normal cellular metabolism
  • 57.
    CAUSES ● Anaphylactic ○ Allergicreaction ● Neurogenic ○ SCI ● Septic ○ Systemic infection ○ Causes release of inflammatory cytokines
  • 58.
    ASSESSMENT ● Decreased oxygen ●Hypotension ● Tachycardia ● Tachypnea ● Warm, flushed skin ● Decreased LOC Specific: ● Anaphylactic ○ Hives ○ Rash ○ Swelling ○ Wheezing ● Neurogenic ○ SCI ○ Priapism ● Septic ○ Hyperthermic ○ Infection
  • 59.
    TREATMENT ● Anaphylactic ○ Epinephrine ○Corticosteroids ○ Bronchodilators ● Neurogenic ○ Cooling ○ Supportive care ● Septic ○ IV antibiotics ○ IVF

Editor's Notes

  • #4 Normal = forward. If blood isn’t moving forward, we have a problem. Red = oxygenated out to the body Blue = deoxygenated returning to heart from body
  • #7 Medications that increase CO Vasodilation to decrease afterload Ace inhibitors - enalapril Arbs - losartan Hydralazine Nitrates Improve contractility Inotropes - dopamine, dobutamine, milrinone Control rate if too fast Beta blockers - propranolol Ccb - verapamil Digoxin Control rhythm if in an arrhythmia Amiodarone
  • #15 H’s and T’s: Hypovolemia Hypoxia Hydrogen Ion excess (Acidosis) Hypo/hyper-kalemia Hypothermia Tension Pneumo Tamponade Thrombosis (MI or PE) Toxins
  • #16 H’s and T’s: Hypovolemia Hypoxia Hydrogen Ion excess (Acidosis) Hypo/hyper-kalemia Hypothermia Tension Pneumo Tamponade Thrombosis (MI or PE) Toxins
  • #17 There is nothing to shock; CPR!!!!
  • #20 Coronary arteries bring blood to the heart muscle - myocardium Only arteries that lead to the heart, not away.
  • #25 Women more likely to have GI symptoms Diabetes less likely to notice symptoms d/t neuropathy Vomiting is due to stimulation of the vagus nerve
  • #27 Morphine helps decrease workload of the heart
  • #34 Fluid retention = heart failure
  • #37 Modifiable vs nonmodifiable
  • #45 1 - loss of adequate blood volume to deliver to tissues = hypovolemic 2 - pump failure = cardiogenic 3 - failure of the vasculature (too much vasodilation) = distributive