CERVICAL SPONDYLOSIS SYNDROME
PRESENATATION BY
DR MISBAHUL FERDOUS
 MBBS(USTC)
 FMD (USTC)
 PGT (CARDIOLOGY) NICVD.DHAKA
PUBLICATION- 1 (ORIGINAL ARTICLE)
METABOLIC SYNDROME AND ACUTE ST ELEVATION MI IN   HOSPITAL
OUTCOME.
PUBLISHED IN B.H.J. JANUARY-2008

MD (CARDIOLOGY), COURSE
SHANDONG UNIVERSITY, CHINA.
Definition
• Degeneration of cervical IVD and the
  secondary degeneration of cervical
  intervertebral joints, leads to injury of
  spinal cord, nerve roots and vertebral
  artery, and shows corresponding
  symptoms and signs
Causes
                                   d e g e n er a io n o f IV D


b u l g e o r e x tr u s i o n o f I V D



                        N a r r o w e d o f i n te r ve r te b r a l s p a c e



                                           l i g a m e n t l ax



                                   u n s tab l e of th e s p i n e



                           h y p e r p l a si a o f v e r te b r a l b o d y,
                                 fa c e t jo i n ts, l i g a m e n ts



                             c o m p r e s s i on to sp i n a l c o r d ,
                                         n e r v e r o o ts,
                                     v e r te b r a l ar te r y
• These accumulated changes caused by
  degeneration can gradually compress
  one or more of the nerve roots.
• This can lead to increasing pain in the
  neck and arm, weakness, and changes in
  sensation.
• In advanced cases, the spinal cord
  becomes involved. This can affect not
  just the arms, but the legs as well.
Causes
• Injury:
    acute injury can further injure
  originally degenerative cervical vertebra
  and discs, this can induce cervical
  spondylosis.
 Chronic injury can speed up process of
  degeneration.
• Congenital deformity:
   stenosis of the cervical spinal canal.
stenosis of the
cervical spinal
canal.



  Pavlov Ratio:
     canal (a) /
  body (b) <0.75
• A previous neck injury (which may have
  occurred several years prior) can
  predispose to spondylosis, but the
  major risk factor is aging.
• By age 60, 70% of women and 85% of
  men show changes consistent with
  cervical spondylosis on X-ray.
As you age, the disks of your spine
become drier and less elastic.
Classification & clinical findings
• Cervical spondylotic radiculopathy (CSR)

• Cervical spondylotic myelopathy (CSM)

• Vertebral artery type of cervical
  spondylopathy
Pathology of CSR
• Most common in morbidity (50%-60%)

• Posterolateral protrusion of the cervical
  disc
• Hyperplasia, hypertrophy of the facet joint .

• stimulate or compress nerve roots as they
  emerge from the cord to pass peripherally
  through the intervertebral foramen
brachial plexus
pathology
Clinical manifestation
• Symptoms
  – Neck pain: radiating to the ipsilateral upper
    extremity
  – Paresthesia
  – Muscle weakness in appropriate distribution

  pain and paresthesia may be intensified by neck
   movement, especially by extension or lateral
   flexion to the side of herniation. May be
   improved by traction on neck.
• Signs
  –Stiffness of neck
  –Tenderness, spasm of paraspinous
   muscles
  –Limitation of active and passive
   motion of the neck and affected
   upper extremity.
Radiographic study


                Demonstrate
                osteophyte
                formation and
                narrowing of
                intervertebral
                foramen.
CT
scan
Pathology-CSM
• Midline herniation of nucleus pulposus
• Osteophyte of posterior rims of vertebral
  body
• Hyperplasia of the ligamentum flavum
• Calcification of the posterior longitudinal
  ligament

Lead to compression of the spinal cord
Clinical manifestation
• Symptoms                  – Numbness
  – Weakness                – Dysfunction of
  – Loss of balance            upper motor neuron
  – Cannot handling small     is gradually present
    objects                   from the lower part
  – Neck pain not obvious     of body to the upper.
                            – Spastic paraplegia
                               or quadriplegia
                            -loss of control of the
                              bladder or bowels
Signs
• Marked motor             • Pyramidal tract sign
  changes and                – Hoffmann’s sign
  relatively few             – Babinski’s sign
  sensory changes.           – Obstacle of fine
  – Hypertonic (high           motion of the fingers
    muscular tone)            Such as buttons,
  – hyperreflexia              write
  – Patellar clonus +
  – Ankle clonus +
Calcification of the posterior
    longitudinal ligament
MRI
Vertebral artery type of CS
• Pathology
• Hyperplasia, stenosis of cervical
  vertebral transverse foramen,
  hypertrophy of upper articular process,
  unstable cervical vertebra
• Directly stimulate, compress or pull
  vertebral artery
Pathology
• Symptoms
  –Vertigo is main, induced by rotating
   neck
  –Migraine
  –Sudden blackout, Diplopia, recovered
   in short time
  –Cataplexy caused by sudden spasm of
   artery due to stimulation, come to at
   once after falling to the ground

• Sign
  –Positive neck rotation test
Treatment
• The goal of treatment is relief of pain
  and prevention of permanent spinal
  cord and nerve root injury.

• In mild cases, no treatment is required.
  Symptoms from cervical spondylosis
  usually stabilize or regress with simple,
  conservative therapy including a neck
  brace and NSAIDs.
Nonoperative treatment
•   Halter traction
•   Cervical support and collar
•   Massage
•   Physical therapy
•   Analgesics and muscle relaxants
•   Local block
Halter traction
• Cervical collar
• Analgesics
  – NSAIDS
    • VOLTAREN
    • Tramcontin
• Muscle relaxants
Operative treatment
• Anterior cervical decompression and
  fusion ( ACDF)
• Artificial disc replacement (ADR)
• Laminectomy
• Laminoplasty
Artificial disc replacement
            (ADR)
Posterior approach
• Indications
• Multiple level spondylosis and diffuse
  spinal canal stenosis.

• Methods
Laminectomy or laminplasty.
Cervical Spondylosis Prevention


• Many cases are not preventable.
  Prevention of neck injury (such as
  proper equipment and techniques when
  playing sports) may reduce risk.
The END!
               Thank You!


                                            !
   Oh, sorry, not the END, just the beginning




Email: misbahul_ferdous@yahoo.com
house no: 26. house name:TAKHDIR.
SUGANDHA. R/A ,CHITTAGONG
BANGLADESH                                      46

Cervical Spondylosis Syndrome

  • 1.
  • 2.
    PRESENATATION BY DR MISBAHULFERDOUS MBBS(USTC) FMD (USTC) PGT (CARDIOLOGY) NICVD.DHAKA PUBLICATION- 1 (ORIGINAL ARTICLE) METABOLIC SYNDROME AND ACUTE ST ELEVATION MI IN HOSPITAL OUTCOME. PUBLISHED IN B.H.J. JANUARY-2008 MD (CARDIOLOGY), COURSE SHANDONG UNIVERSITY, CHINA.
  • 3.
    Definition • Degeneration ofcervical IVD and the secondary degeneration of cervical intervertebral joints, leads to injury of spinal cord, nerve roots and vertebral artery, and shows corresponding symptoms and signs
  • 4.
    Causes d e g e n er a io n o f IV D b u l g e o r e x tr u s i o n o f I V D N a r r o w e d o f i n te r ve r te b r a l s p a c e l i g a m e n t l ax u n s tab l e of th e s p i n e h y p e r p l a si a o f v e r te b r a l b o d y, fa c e t jo i n ts, l i g a m e n ts c o m p r e s s i on to sp i n a l c o r d , n e r v e r o o ts, v e r te b r a l ar te r y
  • 5.
    • These accumulatedchanges caused by degeneration can gradually compress one or more of the nerve roots. • This can lead to increasing pain in the neck and arm, weakness, and changes in sensation. • In advanced cases, the spinal cord becomes involved. This can affect not just the arms, but the legs as well.
  • 6.
    Causes • Injury: acute injury can further injure originally degenerative cervical vertebra and discs, this can induce cervical spondylosis. Chronic injury can speed up process of degeneration. • Congenital deformity: stenosis of the cervical spinal canal.
  • 7.
    stenosis of the cervicalspinal canal. Pavlov Ratio: canal (a) / body (b) <0.75
  • 10.
    • A previousneck injury (which may have occurred several years prior) can predispose to spondylosis, but the major risk factor is aging. • By age 60, 70% of women and 85% of men show changes consistent with cervical spondylosis on X-ray.
  • 11.
    As you age,the disks of your spine become drier and less elastic.
  • 12.
    Classification & clinicalfindings • Cervical spondylotic radiculopathy (CSR) • Cervical spondylotic myelopathy (CSM) • Vertebral artery type of cervical spondylopathy
  • 13.
    Pathology of CSR •Most common in morbidity (50%-60%) • Posterolateral protrusion of the cervical disc • Hyperplasia, hypertrophy of the facet joint . • stimulate or compress nerve roots as they emerge from the cord to pass peripherally through the intervertebral foramen
  • 15.
  • 16.
  • 17.
    Clinical manifestation • Symptoms – Neck pain: radiating to the ipsilateral upper extremity – Paresthesia – Muscle weakness in appropriate distribution pain and paresthesia may be intensified by neck movement, especially by extension or lateral flexion to the side of herniation. May be improved by traction on neck.
  • 18.
    • Signs –Stiffness of neck –Tenderness, spasm of paraspinous muscles –Limitation of active and passive motion of the neck and affected upper extremity.
  • 19.
    Radiographic study Demonstrate osteophyte formation and narrowing of intervertebral foramen.
  • 20.
  • 21.
    Pathology-CSM • Midline herniationof nucleus pulposus • Osteophyte of posterior rims of vertebral body • Hyperplasia of the ligamentum flavum • Calcification of the posterior longitudinal ligament Lead to compression of the spinal cord
  • 22.
    Clinical manifestation • Symptoms – Numbness – Weakness – Dysfunction of – Loss of balance upper motor neuron – Cannot handling small is gradually present objects from the lower part – Neck pain not obvious of body to the upper. – Spastic paraplegia or quadriplegia -loss of control of the bladder or bowels
  • 23.
    Signs • Marked motor • Pyramidal tract sign changes and – Hoffmann’s sign relatively few – Babinski’s sign sensory changes. – Obstacle of fine – Hypertonic (high motion of the fingers muscular tone) Such as buttons, – hyperreflexia write – Patellar clonus + – Ankle clonus +
  • 24.
    Calcification of theposterior longitudinal ligament
  • 25.
  • 26.
    Vertebral artery typeof CS • Pathology • Hyperplasia, stenosis of cervical vertebral transverse foramen, hypertrophy of upper articular process, unstable cervical vertebra • Directly stimulate, compress or pull vertebral artery
  • 27.
  • 28.
    • Symptoms –Vertigo is main, induced by rotating neck –Migraine –Sudden blackout, Diplopia, recovered in short time –Cataplexy caused by sudden spasm of artery due to stimulation, come to at once after falling to the ground • Sign –Positive neck rotation test
  • 29.
  • 30.
    • The goalof treatment is relief of pain and prevention of permanent spinal cord and nerve root injury. • In mild cases, no treatment is required. Symptoms from cervical spondylosis usually stabilize or regress with simple, conservative therapy including a neck brace and NSAIDs.
  • 31.
    Nonoperative treatment • Halter traction • Cervical support and collar • Massage • Physical therapy • Analgesics and muscle relaxants • Local block
  • 32.
  • 34.
  • 37.
    • Analgesics – NSAIDS • VOLTAREN • Tramcontin • Muscle relaxants
  • 38.
    Operative treatment • Anteriorcervical decompression and fusion ( ACDF) • Artificial disc replacement (ADR) • Laminectomy • Laminoplasty
  • 39.
  • 42.
    Posterior approach • Indications •Multiple level spondylosis and diffuse spinal canal stenosis. • Methods Laminectomy or laminplasty.
  • 44.
    Cervical Spondylosis Prevention •Many cases are not preventable. Prevention of neck injury (such as proper equipment and techniques when playing sports) may reduce risk.
  • 46.
    The END! Thank You! ! Oh, sorry, not the END, just the beginning Email: [email protected] house no: 26. house name:TAKHDIR. SUGANDHA. R/A ,CHITTAGONG BANGLADESH 46