DEPARTMENT OF PEDODONTICS

SUBMITTED BY:
SHAYONI SEN
BDS IVTH YEAR



Introduction
Early childhood caries
 Classification



Nursing caries










Etiological agents in nursing bottle caries
Clinical features
Progression of the lesion
Implications
Management
Prevention

Nursing vs rampant caries
Reference


DEFINITION (SHAFER)



Dental caries is an
irreversible microbial
disease of calcified
tissues of the teeth,
characterized by
demineralization of
inorganic portion and
destruction of organic
substance of tooth,
which often leads to
cavitation.
BASED ON
ANATOMIC
SITE
• Occlusal
caries
• Smooth
surface
caries
• Root caries

BASED ON
SEVERITY
• Incipient
caries
• Occult caries
• Cavitation

BASED ON
PROGRESS
ION

BASED ON
CHRONOLOG
Y

• Arrested caries
• Recurrent/
secondary
caries
• Radiation
caries

• Early childhood
caries
• Teenage caries
• Adult caries

CLASSIFICATION
(SHOBHA TANDON)
FLUORID
E
In water

Toothpaste

Varnish
gels

In foods

DIET
Improved
nutrition
Decrease
d amount
of sugar
Decrease
frequency
of sugar

PLAQUE
CONTROL
Better home
care
Better
professional
care
Chemical
control

Antibiotics
In school

Antimicrobial
additives

Changes in
micro flora

SALIV
A

DENTIST/DENTAL
MATERIALS

Changes in
properties

Better
dentists

Secretion
rate

Better
dental
materials

Buffer
capacity

Fissure
sealants

Immunoglobulin
content
Agglutinins
volume



DEFINITION: DAVIES,
1988
A complex disease
involving maxillary
primary incisors within
a month after eruption
and spread rapidly to
involve other primary
teeth.
TYPE 1
ECC
(mild to
moderat
e)

• Carious lesions involving the molars and incisors
• Seen in 2-5 years of age
• Cause is usually a combination of cariogenic semisolid
or solid food and lack of oral hygiene
• Number of affected teeth usually increases as the
cariogenic challenge persists

TYPE 2
ECC
(modera
te to
severe)

• Labiolingual carious lesion affecting the maxillary incisors
with or without molar caries
• Seen soon after 1st tooth erupt
• Cause is inappropriate use of feeding bottle, at-will breast
feeding or combination, poor oral hygiene

TYPE 3
ECC
(severe)

• Carious lesion involve all the teeth, including mandibular
incisors.
• Usually seen in 3-5 years of age
• Cause is combination of factors and poor oral hygiene
• Rampant in nature and involves immune tooth surfaces.




Winter et al, 1966
A unique pattern of dental decay in young
children due to prolonged and improper
nursing/feeding habit.
Etiological
agents in
nursing bottle
caries

Pathogenic
microorganisms

Substrate
(fermentable
carbohydrates)

Host

Time

Other
predisposing
factors








Steptococcus mutans- main
microbe that colonizes teeth
after it erupts into oral cavity.
It is transmitted to infant’s mouth
through mother.
It is more virulent because:It colonizes the teeth
It produces large amount of acid
It produces large amount of
extracellular polysaccharides
that favor plaque formation.
Carbohydrates are converted into dextrans
by microorganisms.
 In infants & toddlers, the main sources of
fermentable carbohydrates are:
i. Bovine milk or infant formulas
ii. Human milk (breast-feeding at will)
iii. Fruit juices & other sweet liquids
iv. Sweet syrups like vitamin preparations
v. Pacifiers dipped in honey or sugar solution
vi. Chocolates or other sweets

Teeth act as host for
microorganisms
 Hypomineralisation or
hypoplasia of teeth
increases the susceptibility
of child to caries
 Thin enamel in primary
teeth is one of the reasons
for early spread of lesions
 Developmental grooves
also may act as plaque
retentive areas



More the time child sleeps with bottle in the
mouth the higher is the risk of caries
because the salivary flow and the swallowing
reflex decrease, thus providing more time for
accumulation of carbohydrates in the mouth
which are acted upon by microbes to
produce acid leading to caries.







Overindulgence of parents
Crowded homes
Child who has less sleep
Malnutrition
Iron deficiency & excess lead exposuresalivary gland function impaired
Low weight infants (<2500 gms)


The intraoral decay pattern is characteristic &
pathognomonic of this condition.

Maxillary central incisors: facial, lingual, mesial, distal surfaces
Maxillary lateral incisors: facial, lingual, mesial, distal surfaces
Maxillary 1st molars: facial, lingual, occlusal, proximal surfaces

Maxillary canines & 2nd molars: facial, lingual, proximal surfaces
Mandibular molars: at later stage


I.

II.

Mandibular anterior
teeth are usually spared
because of:
Protection by tongue
Cleansing action of
saliva due to presence
of the orifice of the duct
of sublingual glands
very close to lower
incisors.
Initially, a
demineralization dull,
white area is seen along
the gum line on labial
aspect of maxillary
incisors.

Finally, the whole
crown of the incisors is
destroyed leaving
behind brown-black
root stumps.

These white lesions
become cavities which
involve the neck of the
tooth in a ring like
fashion





The child who has nursing caries has an
increased risk of developing caries even
in permanent dentition.
The child with caries is also susceptible to
other heath hazards.
The treatment of nursing caries may prove
to be financial burden for some parents.
 Aims:
I. Management of existing emergency
II. Arrest & control of the carious process
III. Institution of preventive procedure
IV. Restoration & rehabilitation
 Factors affecting management:
I. Extent of the lesion
II. Age of the patient
III. Behavioral problems due to young age

the child

of
 All

lesions should be excavated and
restored
 Indirect pulp capping or pulp therapy
procedures can be evaluated by further
investigation
 If the abscess is present it can be treated
by drainage
 X-Rays are advised to assess the
condition of succedaneous teeth collection
of saliva for determining the salivary flow
& viscosity
Parent should be questioned about the child’s
feeding habits, nocturnal bottles, demand for
breast-feeding, pacifiers.
 Parents should be asked to try weaning the child
from using the bottle as pacifier while in bed.
 In case of emotional dependence on the bottle,
suggest use of plain or fluoridated water.
 The parents should be instructed to clean the
child’s teeth after every feed.
 Parents are advised to maintain a diet record of
the child for 1 week that includes the time, amount
of food given to the child, the type of the food &
the number of sugar exposures.

Should be scheduled 1 week after 1st week.
 Analysis of diet chart & explanation of
disease process of child’s teeth
 Isolate the sugar factors from diet chart &
control sugar exposure
 Reassess the restoration and redo if
needed
 Caries activity tests can be started &
repeated at monthly interval to monitor the
success of treatment






Restoring all grossly decayed teeth
Endodontic treatment
In case of unrestorable teeth, extraction
followed by space maintainer
Crowns given for grossly decayed &
endodontically treated teeth
Review & recall after every 3 months







Information of nursing bottle caries can be
distributed to new parents through
obstetricians, pediatricians & child care
centers.
Sealing of all pits & fissure caries
Professional fluoride programs
Use of antimicrobial therapy topically
Systemic fluoride in drinking water
NURSING CARIES
Specific form of rampant caries.

RAMPANT CARIES
Acute, widespread caries with early pulpal
involvement of teeth which are usually
immune to decay.

Age of occurrence
Age of occurrence
In infants & toddlers
Seen at all ages
Dentition involved
Affects the primary dentition
Characteristic feature

Specific pattern is seen
Mandibular molars are not involved

Dentition involved
Both primary & permanent dentition
Characteristic feature

Surfaces considered immune to decay are
involved. Thus, mandibular incisors are
affected
NURSING CARIES

RAMPANT CARIES

Etiology

Etiology

Bottle feeding before sleep

More multifactorial with all the essential factors
involved are not just feeding practices

Pacifiers dipped in honey

Frequent snacks, excessive sticky refined
carbohydrates
Decreased salivary flow

Prolonged at-will beast-feeding
Genetic background

Treatment
Treatment

In early stage- topical fluoride application &
education

Require pulp therapy

Directed toward maintenance of the teeth till
the transition occurs

Long term treatment

Prevention

Prevention

Education of the parents

Dental Health Education at a mass level involving
people of all ages


SHOBHA TONDON (FOR PEDIATRICS
DENTISTRY) 2nd EDITION.
Early childhood caries

Early childhood caries

  • 1.
    DEPARTMENT OF PEDODONTICS SUBMITTEDBY: SHAYONI SEN BDS IVTH YEAR
  • 2.
      Introduction Early childhood caries Classification  Nursing caries         Etiological agents in nursing bottle caries Clinical features Progression of the lesion Implications Management Prevention Nursing vs rampant caries Reference
  • 3.
     DEFINITION (SHAFER)  Dental cariesis an irreversible microbial disease of calcified tissues of the teeth, characterized by demineralization of inorganic portion and destruction of organic substance of tooth, which often leads to cavitation.
  • 4.
    BASED ON ANATOMIC SITE • Occlusal caries •Smooth surface caries • Root caries BASED ON SEVERITY • Incipient caries • Occult caries • Cavitation BASED ON PROGRESS ION BASED ON CHRONOLOG Y • Arrested caries • Recurrent/ secondary caries • Radiation caries • Early childhood caries • Teenage caries • Adult caries CLASSIFICATION (SHOBHA TANDON)
  • 5.
    FLUORID E In water Toothpaste Varnish gels In foods DIET Improved nutrition Decrease damount of sugar Decrease frequency of sugar PLAQUE CONTROL Better home care Better professional care Chemical control Antibiotics In school Antimicrobial additives Changes in micro flora SALIV A DENTIST/DENTAL MATERIALS Changes in properties Better dentists Secretion rate Better dental materials Buffer capacity Fissure sealants Immunoglobulin content Agglutinins volume
  • 6.
      DEFINITION: DAVIES, 1988 A complexdisease involving maxillary primary incisors within a month after eruption and spread rapidly to involve other primary teeth.
  • 7.
    TYPE 1 ECC (mild to moderat e) •Carious lesions involving the molars and incisors • Seen in 2-5 years of age • Cause is usually a combination of cariogenic semisolid or solid food and lack of oral hygiene • Number of affected teeth usually increases as the cariogenic challenge persists TYPE 2 ECC (modera te to severe) • Labiolingual carious lesion affecting the maxillary incisors with or without molar caries • Seen soon after 1st tooth erupt • Cause is inappropriate use of feeding bottle, at-will breast feeding or combination, poor oral hygiene TYPE 3 ECC (severe) • Carious lesion involve all the teeth, including mandibular incisors. • Usually seen in 3-5 years of age • Cause is combination of factors and poor oral hygiene • Rampant in nature and involves immune tooth surfaces.
  • 8.
      Winter et al,1966 A unique pattern of dental decay in young children due to prolonged and improper nursing/feeding habit.
  • 9.
  • 10.
          Steptococcus mutans- main microbethat colonizes teeth after it erupts into oral cavity. It is transmitted to infant’s mouth through mother. It is more virulent because:It colonizes the teeth It produces large amount of acid It produces large amount of extracellular polysaccharides that favor plaque formation.
  • 11.
    Carbohydrates are convertedinto dextrans by microorganisms.  In infants & toddlers, the main sources of fermentable carbohydrates are: i. Bovine milk or infant formulas ii. Human milk (breast-feeding at will) iii. Fruit juices & other sweet liquids iv. Sweet syrups like vitamin preparations v. Pacifiers dipped in honey or sugar solution vi. Chocolates or other sweets 
  • 12.
    Teeth act ashost for microorganisms  Hypomineralisation or hypoplasia of teeth increases the susceptibility of child to caries  Thin enamel in primary teeth is one of the reasons for early spread of lesions  Developmental grooves also may act as plaque retentive areas 
  • 13.
     More the timechild sleeps with bottle in the mouth the higher is the risk of caries because the salivary flow and the swallowing reflex decrease, thus providing more time for accumulation of carbohydrates in the mouth which are acted upon by microbes to produce acid leading to caries.
  • 14.
          Overindulgence of parents Crowdedhomes Child who has less sleep Malnutrition Iron deficiency & excess lead exposuresalivary gland function impaired Low weight infants (<2500 gms)
  • 15.
     The intraoral decaypattern is characteristic & pathognomonic of this condition. Maxillary central incisors: facial, lingual, mesial, distal surfaces Maxillary lateral incisors: facial, lingual, mesial, distal surfaces Maxillary 1st molars: facial, lingual, occlusal, proximal surfaces Maxillary canines & 2nd molars: facial, lingual, proximal surfaces Mandibular molars: at later stage
  • 16.
     I. II. Mandibular anterior teeth areusually spared because of: Protection by tongue Cleansing action of saliva due to presence of the orifice of the duct of sublingual glands very close to lower incisors.
  • 17.
    Initially, a demineralization dull, whitearea is seen along the gum line on labial aspect of maxillary incisors. Finally, the whole crown of the incisors is destroyed leaving behind brown-black root stumps. These white lesions become cavities which involve the neck of the tooth in a ring like fashion
  • 18.
       The child whohas nursing caries has an increased risk of developing caries even in permanent dentition. The child with caries is also susceptible to other heath hazards. The treatment of nursing caries may prove to be financial burden for some parents.
  • 19.
     Aims: I. Managementof existing emergency II. Arrest & control of the carious process III. Institution of preventive procedure IV. Restoration & rehabilitation  Factors affecting management: I. Extent of the lesion II. Age of the patient III. Behavioral problems due to young age the child of
  • 20.
     All lesions shouldbe excavated and restored  Indirect pulp capping or pulp therapy procedures can be evaluated by further investigation  If the abscess is present it can be treated by drainage  X-Rays are advised to assess the condition of succedaneous teeth collection of saliva for determining the salivary flow & viscosity
  • 21.
    Parent should bequestioned about the child’s feeding habits, nocturnal bottles, demand for breast-feeding, pacifiers.  Parents should be asked to try weaning the child from using the bottle as pacifier while in bed.  In case of emotional dependence on the bottle, suggest use of plain or fluoridated water.  The parents should be instructed to clean the child’s teeth after every feed.  Parents are advised to maintain a diet record of the child for 1 week that includes the time, amount of food given to the child, the type of the food & the number of sugar exposures. 
  • 22.
    Should be scheduled1 week after 1st week.  Analysis of diet chart & explanation of disease process of child’s teeth  Isolate the sugar factors from diet chart & control sugar exposure  Reassess the restoration and redo if needed  Caries activity tests can be started & repeated at monthly interval to monitor the success of treatment
  • 23.
         Restoring all grosslydecayed teeth Endodontic treatment In case of unrestorable teeth, extraction followed by space maintainer Crowns given for grossly decayed & endodontically treated teeth Review & recall after every 3 months
  • 24.
         Information of nursingbottle caries can be distributed to new parents through obstetricians, pediatricians & child care centers. Sealing of all pits & fissure caries Professional fluoride programs Use of antimicrobial therapy topically Systemic fluoride in drinking water
  • 26.
    NURSING CARIES Specific formof rampant caries. RAMPANT CARIES Acute, widespread caries with early pulpal involvement of teeth which are usually immune to decay. Age of occurrence Age of occurrence In infants & toddlers Seen at all ages Dentition involved Affects the primary dentition Characteristic feature Specific pattern is seen Mandibular molars are not involved Dentition involved Both primary & permanent dentition Characteristic feature Surfaces considered immune to decay are involved. Thus, mandibular incisors are affected
  • 27.
    NURSING CARIES RAMPANT CARIES Etiology Etiology Bottlefeeding before sleep More multifactorial with all the essential factors involved are not just feeding practices Pacifiers dipped in honey Frequent snacks, excessive sticky refined carbohydrates Decreased salivary flow Prolonged at-will beast-feeding Genetic background Treatment Treatment In early stage- topical fluoride application & education Require pulp therapy Directed toward maintenance of the teeth till the transition occurs Long term treatment Prevention Prevention Education of the parents Dental Health Education at a mass level involving people of all ages
  • 28.
     SHOBHA TONDON (FORPEDIATRICS DENTISTRY) 2nd EDITION.