CONCOMITANT STRABISMUS
Dr. Ritu Chaturvedi
HOD, Department of Ophthalmology
SRVS, Medical College
A type of manifest
squint in which the
amount of deviation in
the squinting eye
remains constant in all
directions of gaze; and
there is no associated
limitation of ocular
movements
ETIOLOG
Y
• Binocular vision and coordination of ocular
movements are not present since birth but are
acquired in the early childhood.
• The process starts by the age of 3-6 months and
is completed up to 5-6 years. Therefore, any
obstacle to the development of these processes
may result in concomitant squint.
ETIOLOG
Y
Sensory obstacles
• Refractive errors
• Prolonged use of
incorrect spectacles
• Anisometropia
• Corneal opacities
• Lenticular opacities
• Diseases of macula
• Optic atrophy
• Obstruction in the
pupillary area due to
congenital ptosis
Motor obstacles
• Congenital
abnormalities of the
shape and size of the
orbit
• Abnormalities of
extraocular muscles
• Abnormalities of
accommodation,
convergence and AC/A
ratio
Central obstacles
• Deficient development
of fusion faculty
• Abnormalities of cortical
control of ocular
movements, and
hyperexcitability of the
CNS during teething
CLINICAL
FEATURES
IN GENERAL
1. OCULAR DEVIATION
• Unilateral or alternating
• Inward deviation or outward deviation or vertical deviation
• Primary deviation is equal to secondary deviation
• Ocular deviation is equal in all directions of gaze
2. OCULAR MOVEMENT
• Not limited in any direction
3. REFRACTIVE ERROR
• May or may not be associated
4. SUPPRESSION AND AMBLYOPIA
• May be develop as sensory adaptation to strabismus
• Amblyopia develops in monocular strabismus only and is
responsible for poor visual acuity
5. A-V PATTERNS
• May be observed in horizontal strabismus.
• when this patterns associated, the horizontal concomitant
strabismus becomes vertically incomitant
V esotropia
A esotropia
Convergent
squint
(esotropia)
Divergent
squint
(exotropia)
Vertical
squint
(hypertropia)
TYPE
S
• Denotes inward deviation of one eye and is the
most common type of squint in children.
• Unilateral or alternating
CONVERGENT
SQUINT
1. INFANTILE ESOTROPIA
• Age of onset, usually 1-2 months, but may occur during first 6 months
of life
• Angle of deviation is constant and fairly large (>30 degree)
• Fixation pattern
• Binocular vision does not develop and there is alternate fixation in
primary gaze and cross fixation in the lateral gaze
• Amblyopia in 25-40% cases
• Treatment
• Amblyopia treatment by patching the normal eye should always be
done before performing surgery
• Recession of both medial recti is preferred over unilateral
recess-resect procedure
• Surgery should be done between 6 months – 2 years; preferably <1
year
2. ACCOMMODATIVE ESOTROPIA
• Occurs due to overaction of convergence associated with accommodation reflex
• 3 types
• Refractive accommodative esotropia
• Associated with high hypermetropia (+4 to +7D)
• Fully correctable by use of spectacles
• Non-refractive accommodative esotropia
• Caused by AC/A ratio
• Esotropia is greater for near than that for distance
• Fully corrected by bifocal glasses with add +3DS for near vision
• Mixed accommodative esotropia
• Caused by combination of hypermetropia and high AC/A ratio
• Esotropia for distance is corrected by correction of hypermetropia; and
the residual esotropia for near is corrected by addition of +3DS lens
3. ACQUIRED NON-ACCOMMODATIVE ESOTROPIAS
• Includes all those acquired primary esodeviations in which amount of
deviation is not affected by the state of accommodation
4. SENSORY ESOTROPIA
• Results from monocular lesions in childhood which either prevent the
development of normal binocular vision or interfere with its
maintenance
5. CONSECUTIVE ESOTROPIA
• Result from surgical overcorrection of exotropia
• Characterised by outward deviation of
one eye while the other eye fixates
DIVERGENT
SQUINT
Types
– Congenital exotropia
– Primary exotropia
– Secondary exotropia
– Consecutive exotropia
Rare, almost present at birth
EVALUATION
• History
• Examination:
- inspection
- ocular movements
- pupillary reactions
- media & fundus examination
- testing of vision & refractive
error
- cover tests (direct and alternate)
- estimation of angle of deviation
- tests for grade of binocular
vision and sensory functions
• confirms the presence of
manifest squint
ii.
iii.
iv.
TREATMENT
• Goals of treatments:
- To achieve good cosmetic correction
- To improve visual acuity
- To maintain binocular single vision
• Treatment modalities:
- spectacles with full correction of refractive error
- occlusion therapy
- preoperative orthoptic exercises
- squint surgery
- postoperative orthoptic exercises
• Squint surgery
– Should always be instituted after the
correction of refractive error, treatment of
amblyopia and orthoptic exercises.
 Basic principles:
 These are to weaken the strong
muscle by recession (shifting the
insertion posteriorly) or to strengthen
the weak muscle by resection
(shortening the muscle).
thank you

Introduction to Comitant strabismus.pptx

  • 1.
    CONCOMITANT STRABISMUS Dr. RituChaturvedi HOD, Department of Ophthalmology SRVS, Medical College
  • 2.
    A type ofmanifest squint in which the amount of deviation in the squinting eye remains constant in all directions of gaze; and there is no associated limitation of ocular movements
  • 3.
    ETIOLOG Y • Binocular visionand coordination of ocular movements are not present since birth but are acquired in the early childhood. • The process starts by the age of 3-6 months and is completed up to 5-6 years. Therefore, any obstacle to the development of these processes may result in concomitant squint.
  • 4.
    ETIOLOG Y Sensory obstacles • Refractiveerrors • Prolonged use of incorrect spectacles • Anisometropia • Corneal opacities • Lenticular opacities • Diseases of macula • Optic atrophy • Obstruction in the pupillary area due to congenital ptosis Motor obstacles • Congenital abnormalities of the shape and size of the orbit • Abnormalities of extraocular muscles • Abnormalities of accommodation, convergence and AC/A ratio Central obstacles • Deficient development of fusion faculty • Abnormalities of cortical control of ocular movements, and hyperexcitability of the CNS during teething
  • 5.
    CLINICAL FEATURES IN GENERAL 1. OCULARDEVIATION • Unilateral or alternating • Inward deviation or outward deviation or vertical deviation • Primary deviation is equal to secondary deviation • Ocular deviation is equal in all directions of gaze
  • 6.
    2. OCULAR MOVEMENT •Not limited in any direction 3. REFRACTIVE ERROR • May or may not be associated 4. SUPPRESSION AND AMBLYOPIA • May be develop as sensory adaptation to strabismus • Amblyopia develops in monocular strabismus only and is responsible for poor visual acuity 5. A-V PATTERNS • May be observed in horizontal strabismus. • when this patterns associated, the horizontal concomitant strabismus becomes vertically incomitant
  • 7.
  • 8.
  • 9.
    • Denotes inwarddeviation of one eye and is the most common type of squint in children. • Unilateral or alternating CONVERGENT SQUINT
  • 10.
    1. INFANTILE ESOTROPIA •Age of onset, usually 1-2 months, but may occur during first 6 months of life • Angle of deviation is constant and fairly large (>30 degree) • Fixation pattern • Binocular vision does not develop and there is alternate fixation in primary gaze and cross fixation in the lateral gaze • Amblyopia in 25-40% cases • Treatment • Amblyopia treatment by patching the normal eye should always be done before performing surgery • Recession of both medial recti is preferred over unilateral recess-resect procedure • Surgery should be done between 6 months – 2 years; preferably <1 year
  • 11.
    2. ACCOMMODATIVE ESOTROPIA •Occurs due to overaction of convergence associated with accommodation reflex • 3 types • Refractive accommodative esotropia • Associated with high hypermetropia (+4 to +7D) • Fully correctable by use of spectacles • Non-refractive accommodative esotropia • Caused by AC/A ratio • Esotropia is greater for near than that for distance • Fully corrected by bifocal glasses with add +3DS for near vision • Mixed accommodative esotropia • Caused by combination of hypermetropia and high AC/A ratio • Esotropia for distance is corrected by correction of hypermetropia; and the residual esotropia for near is corrected by addition of +3DS lens
  • 12.
    3. ACQUIRED NON-ACCOMMODATIVEESOTROPIAS • Includes all those acquired primary esodeviations in which amount of deviation is not affected by the state of accommodation 4. SENSORY ESOTROPIA • Results from monocular lesions in childhood which either prevent the development of normal binocular vision or interfere with its maintenance 5. CONSECUTIVE ESOTROPIA • Result from surgical overcorrection of exotropia
  • 13.
    • Characterised byoutward deviation of one eye while the other eye fixates DIVERGENT SQUINT Types – Congenital exotropia – Primary exotropia – Secondary exotropia – Consecutive exotropia Rare, almost present at birth
  • 14.
    EVALUATION • History • Examination: -inspection - ocular movements - pupillary reactions - media & fundus examination - testing of vision & refractive error - cover tests (direct and alternate) - estimation of angle of deviation - tests for grade of binocular vision and sensory functions • confirms the presence of manifest squint ii. iii. iv.
  • 17.
    TREATMENT • Goals oftreatments: - To achieve good cosmetic correction - To improve visual acuity - To maintain binocular single vision • Treatment modalities: - spectacles with full correction of refractive error - occlusion therapy - preoperative orthoptic exercises - squint surgery - postoperative orthoptic exercises
  • 18.
    • Squint surgery –Should always be instituted after the correction of refractive error, treatment of amblyopia and orthoptic exercises.  Basic principles:  These are to weaken the strong muscle by recession (shifting the insertion posteriorly) or to strengthen the weak muscle by resection (shortening the muscle).
  • 20.