POLYHYDRAMNIOS
•Definition:
• Polyhydramnios is defined as a state where liquor amnii exceeds 2000 ml or when A.F.I.
is more than 24-25 cm or a single pocket of amniotic fluid is greater than 8 cm by
ultrasonography.
• Incidence: 1% to 2 % of the cases
• Causes
• Maternal (15%)
Rh iso-immunization
DM
• Placental (less than 1%)
Placental chorioangioma
Circumvallate placental syndrome
• Fetal (18%)
Multiple pregnancies
Fetal anomalies
• Idiopathic (65%)
• Clinical types: Depending on the rapidity of onset
hydramnios can be
Acute – rare – appear in a matter of few
days
Chronic – more common 10 times more
commoner to acute appear in a matter of few
months
• Routine OBH
• History suggestive of Rh iso- immunization such as still birth, fetal hydrops,
jaundice in new born requiring exchange transfusion etc.
• History suggestive of DM – Previous big baby fetal death at 35 weeks,
classical symptoms of DM like polyurea, polydypsia, polyphagia
• History of Drug intake especially in First trimester
• History of Previous fetal anomalies like Anencephaly-risk of recurrence is
2%
• Acute Polyhydramnios: Onset is acute usually occurs before
20 weeks of pregnancy and presents usually with symptoms and labour starts
before 28 weeks of pregnancy.
• It may present as
Acute abdomen - abdominal pain, nausea, vomiting
Breathlessness which increases on lying down position
Palpitation
Oedema of legs, varicosities in legs, vulva and hemorroids
• Signs:
Patient looks ill, with out features of shock
Oedema of legs with signs of PIH
Abdomen unduly enlarged with shiny skin
Fluid thrill may be present
• Internal examination shows taking up of cervix or even dilatation with bulging
membranes
• Chronic Polyhydramnios: More common than
acute 10% more common
• Since accumulation of liquor is gradual and so patient may be
symptomatic or asymptomatic.
• Symptomsare mainly due to mechanical causes
Dyspnoea is more in supine position
Palpitation
Oedema
Oliguria may result from ureteral obstruction by enlarged
uterus
• Pre-eclampsia 25 %( oedema, hypertension and proteinuria)
Signs GPE
• Patient may be dyspnoic at rest
• Pedal Oedema
• Evidence of PIH
Abdominal examination
Inspection
• Abdomen is markedly enlarged globular with fullness in flanks
• Skin over the abdomen is tense shiny with large striae
Palpation
• Height of uterus is more than the corresponding periods of Amenorrhoea
• Abdominal girth is more
• Fetal parts cannot be well defined external ballotment is more easily elicited
• Malpresentations are more common and presenting part is usually high up
• Fluid thrill is present
Auscultation
• Fetal heart sounds are not heard distinctly
Internal examination :
Cervix is pulled up
May be sometimes dilated and admits tip of finger through
which bag of membranes which is tense is felt.
• At times patient may present with complicationslike
Pre ecclampsia
PROM
Preterm labour
Placental abruption
Cord prolapse
Polyhydramnios- Define, Incidence, Causes,Sign and Symptoms, Diagnosis, types,General examination PPT

Polyhydramnios- Define, Incidence, Causes,Sign and Symptoms, Diagnosis, types,General examination PPT

  • 1.
  • 2.
    •Definition: • Polyhydramnios isdefined as a state where liquor amnii exceeds 2000 ml or when A.F.I. is more than 24-25 cm or a single pocket of amniotic fluid is greater than 8 cm by ultrasonography. • Incidence: 1% to 2 % of the cases • Causes • Maternal (15%) Rh iso-immunization DM • Placental (less than 1%) Placental chorioangioma Circumvallate placental syndrome • Fetal (18%) Multiple pregnancies Fetal anomalies • Idiopathic (65%)
  • 3.
    • Clinical types:Depending on the rapidity of onset hydramnios can be Acute – rare – appear in a matter of few days Chronic – more common 10 times more commoner to acute appear in a matter of few months
  • 4.
    • Routine OBH •History suggestive of Rh iso- immunization such as still birth, fetal hydrops, jaundice in new born requiring exchange transfusion etc. • History suggestive of DM – Previous big baby fetal death at 35 weeks, classical symptoms of DM like polyurea, polydypsia, polyphagia • History of Drug intake especially in First trimester • History of Previous fetal anomalies like Anencephaly-risk of recurrence is 2%
  • 5.
    • Acute Polyhydramnios:Onset is acute usually occurs before 20 weeks of pregnancy and presents usually with symptoms and labour starts before 28 weeks of pregnancy. • It may present as Acute abdomen - abdominal pain, nausea, vomiting Breathlessness which increases on lying down position Palpitation Oedema of legs, varicosities in legs, vulva and hemorroids • Signs: Patient looks ill, with out features of shock Oedema of legs with signs of PIH Abdomen unduly enlarged with shiny skin Fluid thrill may be present • Internal examination shows taking up of cervix or even dilatation with bulging membranes
  • 6.
    • Chronic Polyhydramnios:More common than acute 10% more common • Since accumulation of liquor is gradual and so patient may be symptomatic or asymptomatic. • Symptomsare mainly due to mechanical causes Dyspnoea is more in supine position Palpitation Oedema Oliguria may result from ureteral obstruction by enlarged uterus • Pre-eclampsia 25 %( oedema, hypertension and proteinuria)
  • 7.
    Signs GPE • Patientmay be dyspnoic at rest • Pedal Oedema • Evidence of PIH Abdominal examination Inspection • Abdomen is markedly enlarged globular with fullness in flanks • Skin over the abdomen is tense shiny with large striae Palpation • Height of uterus is more than the corresponding periods of Amenorrhoea • Abdominal girth is more • Fetal parts cannot be well defined external ballotment is more easily elicited • Malpresentations are more common and presenting part is usually high up • Fluid thrill is present Auscultation • Fetal heart sounds are not heard distinctly
  • 8.
    Internal examination : Cervixis pulled up May be sometimes dilated and admits tip of finger through which bag of membranes which is tense is felt. • At times patient may present with complicationslike Pre ecclampsia PROM Preterm labour Placental abruption Cord prolapse