SURGICAL MANAGEMENT OF PPH
AT TERTIARY CENTER




PROF. (Dr ) M.C. Bansal
MBBS, MS , FICOG , MICOG .
Pregnancy
the most dangerous journey of mankind…
Definition
WHO defines PPH as blood loss of more than 500
ml following vaginal delivery or more than 1000 ml
after caesarean section. In Asian women even loss
of 300 ml can have sinister effect due to smaller
built, lesser blood volume, lower Hb & poor
nutrition. However, various authors suggest that
PPH should be diagnosed with any amount of
blood loss that     threatens the hemodynamic
stability of the woman.
Causes of Maternal Death

                Haemorrhage
                   24.8%

                                   Infection
                                    14.9%
                                                  Haemorrhage
Indirect
                                                  is the biggest
 causes
                                                    and fastest
 19.8%
                                                       killer
                                     Eclampsia
                                      12.9%
Other direct
  causes                      Obstructed labour
                Unsafe
   7.9%                             6.9%
                abortion
                 12.9%
Postpartum Hemorrhage

 PPH is a serious, Life-threatening obstetric problem.

 One of the leading causes of maternal morbidity and
mortality.

 In developing countries mainly due to three delays: -
1. Delay in seeking care.
2. Delay in reaching care.
3. Delay in receiving care.
Maternal Mortality following PPH

WHO estimated
5,29,000 maternal death / year in world
1,36,000 maternal death/ year in India i.e.
 30%
29.6% maternal deaths due of PPH.
In India, about 15.15% - 25.8% mothers die
 due to PPH.
Additional burden of PPH


• > 50% of births in India take place at
  home without the help of trained/
  qualified Birth attendant .

• 83% of rural deliveries occur at home

• 80% of women are anaemic
Incidence of PPH
• 11% women with live birth i.e. 14 million
 women / year
• 3.9% in vaginal deliveries

• 6.4% in Cesarean section .

• Higher with high risk factor

• 10% overall.

• Mismanagement of III stage results in
 higher incidence of PPH
The Four Ts Mnemonic – Causes of PPH

         Four Ts                           Causes            Incidence (%)

       1st Tone                         Atonic uterus             70

              2nd                  Lacerations, hematomas,        20
         Trauma                       inversion, rupture

      3rd Tissue                       Retained tissue,           10
                                      Invasive placenta

              4th                      Coagulopathies             1
      Thrombin
Am Fam Physician 2007;75:875-82.
MMR & MORBIDITY DUE TO OBSTETRICAL
         HAEMORRHAGE
‘Prevention is
easier and better
   than cure’
Prevention of PPH ???
  It can be achieved
             by
 Active management
            of
   3 rd stage of labour
Active Management – Why?

                  30


                  20
        Percent




                  10


                   0
                       Transfusion Prolonged 3rd Therapeutic     Low        Retained
                                       Stage     Uterotonic    Hemoglobin   Placenta

                  Active Management    Physiological Management        McMormick, Sanghvi,
                                                                        Kinzie, McIntosh,
                                                                           IJGO2003


1. Reduces length of time of 3rd stage
2. Reduces amount of blood loss
3. Reduces need for blood transfusions
Prevention
   Universalization of Spancer’s
    Modification of Brand- Andrew’s
    technique of placental delivery e.g.
    prophylactic injection of uterotonics at
    the time of delivery of anterior shoulder
    followed by traction and counter
    traction maneuver for placental
    delivery.
   Prompt recognition and aggressive
    management according to the cause of
    PPH.
Prevention (contd)

  Shout for help 

  1. Blood bank , relative, donors, blood and
        blood components.
  2.   Anesthetist
  3.   Prepare Operation Theater.
  4.   Immediate Communication with nearest/
        dependent Tertiary center,
  5.   Quick transportation to Tertiary Centre
        (summon obstetrical flying squad /108
         Ambulance)
Recognition   Referral   Responsiveness
“No matter where a woman delivers, giving birth
 should be a moment of joy, not a sentence to
                    death”
“While managing PPH
Time lapsed should not
be counted in a minute-
--one has not lost one
minute ,but 60 seconds”
          Ian Donald
“No amount of Blood from any
blood bank is safer and better
than Her own blood.”

       Hence be prompt in
saving each second and every
drop of blood of bleeding woman
in her 3rd stage of labour,
PPH Treatment Protocol
PPH Treatment Protocol
Treatment Protocol Of Primary Atonic PPH
                    (1st T)



  Management                   Management of
  of Shock               Uterine atonicity



Replacement of blood      * Conservative medical     or its
component              management

                           * Surgical management
                            - Conservative surgery
                            - Radical surgery
Stepwise Management of Atonic PPH
Step I - Bleeding continues
        - 15 methyl PGF2 250g every 15-30 mint.
Step II - a) Bimanual compression
        b) Aortic compression
Step III - Transvaginal options
         - Uterine packing
         - Tamponade
Step IV - Compression sutures
       B.Lynch, Hayman, Cho Square
Step V -Other surgical measures
         - stepwise uterine devascularisation

Step VI - Hysterectomy
Conservative Surgical Management


Mode of Actions:

 Controls PPH
 Preserves reproductive functions

 Avoids hysterectomy and related
 complications and consequences
Bimanual Compression of Relaxed Uterus
Conservative Surgical Management
           Options In Atonic PPH
 Manual removal of placenta
 Exploration of uterus
 Uterine Packing by Roller gauze, Condom,
  Foley Catheter, Sengstaken Blackmore tube,
  Surgical         Glove       distention,Balloon
  tamponade
 Step-wise devascularization of uterus
 B-Lynch suture, Hayman suture, Gun Sheela,
   Cho multiple square suture
 Internal iliac (hypogastric) artery ligation
 Postpartum Arterial embolisation
MRP
MRP
UTERINE PACCKING PLASTC BAG INFUSED WITH
                 SALINE
Balloon Tamponade
 Two-way catheter - temporary
 control of PPH
 Feasible in a scenario of atonic
 PPH following a vaginal delivery,
 unresponsive to medical
 management & before
 interventional radiological
 procedures or surgical
 interventions
 Simple, cheap, easy to use &
 effective measure
Procedure

 Balloon portion is placed directly into uterus [entire balloon
  (500ml capacity) has to be inserted past the cervical canal &
  internal os].
 Gentle traction on balloon shaft ensures proper contact
  between balloon & tissue surface & enhances tamponade
  effect
 Success is judged by a declining loss of blood from cervix &
  that seen through drainage port
 Mean time for leaving the tamponade balloon - 8 to 48 hours
 Gradual deflation of the balloon is advised to reduce the
  potential risk of further bleeding
Step-Wise Devascularisation Of The Uterus

 1st reported from Egypt
 Effective in controlling PPH in 80% of cases
 Unilateral uterine artery ligation
 Bilateral uterine artery ligation at the upper part of the
  lower uterine segment
 Low uterine vessels ligation after mobilization of the
  bladder
 Unilateral ovarian vessel ligation
 Bilateral ovarian vessel ligation
Ovarian Artery Ligation


 Ovarian artery directly arises from the aorta

 Anastomosis with the uterine artery in the region of
  the uterine aspect of the utero-ovarian ligament
Uterine Artery Ligation

 90% blood supply of uterus in pregnancy
 is from uterine vessels


 Ligation of uterine arteries result into
 significant reduction in blood flow to the
 uterus
Uterine
 Artery
Ligation

ABDOMINAL
  ROUTE
Vaginal Route for
         Uterine Artery Ligation


 Indicated in

  atonic PPH
  following
 vaginal
 delivery
B-Lynch Suture
 Exerts continuous vertical compression on uterine
 vascular system
 Before proceeding to place the suture into uterus,
 potential efficacy of B-Lynch suture should be tested for
 by performing open bimanual compression to see if
 bleeding stops
 The assistant performs compression & maintains it with
 2 hands during the placement of the suture by the
 surgeon
 Monocryl suture or Vicryl number 2 should be used
B- LYNCH
SUTURE
B-LYNCH SUTURE
B-LYNCH
SUTURE
B-
Lynch
Suture
B-LINCH SUTURE
Cho Multiple Square
          Compression Sutures
 Multiple square sutures
 are used to cover the
 whole body of uterus using
 a straight 10-cm needle
 May be useful in placenta
 previa
Sketch of pelvic blood vessels
Internal Iliac Artery
(Anatomy-Surgical dissection)
INTERNAL ILLIAC ARTERY (ANATOMY) SURGICAL DISSECTION
Internal Iliac Artery Ligation

Conditions indicating ligation –
 Atonic uterus refractory to
 other measures
 Abruptio placentae with uterine
 atony
 Abdominal pregnancy with
 pelvic implantation of the
 placenta & placenta accreta
Internal Iliac Artery Ligation

T   Therapeutic indications
     Before or after hysterectomy for PPH

     Continuous bleeding from the broad ligament base;
     profuse bleeding from pelvic side-wall or vaginal angle
     Diffuse bleeding without , clearly identifiable vascular

     bed
     Ruptured uterus in which uterine artery may be torn at

     its origin from internal iliac artery
     Where extensive lacerations of cervix have occurred
     following difficult instrumental delivery
INTERNAL ILLIAC ARTERY
      LIGATION
Uterine Artery Embolization

 Highly feasible, safe & beneficial procedure,
 possibly precluding further laparotomy &
 hysterectomy
 If successful, not only saves the patient’s life,
 but also preserves the functions of uterus
 ,tubes and ovaries.
 Should be the procedure of choice for PPH
 prior to surgical intervention
ANGIOGRAPHY – UTERINE ARTERY
BLOCKED
 uterine
 ARTERY
   after
EMBOLIZ
  ATION
Hysterectomy
 Best immediate option
   When uterine atony is unresponsive to uterotonics
   Where facilities for embolization are not available
   Obstetrician not well versed with technical aspects of
    conservative surgical procedures or iliac artery ligation

 Indications
   Uterine rupture secondary to obstructed labor
   Previous Caesarean section
   If rupture is extensive & hemorrhage cannot be contained by
    suture of ruptured area
PPH in CASE of ABRUPTIO PLACENTA ( Covouliare Uterus)
2nd T Surgical Treatment of
        Traumatic PPH
Causes 
 1.CervicalTear-Lateral, annular, bucket handle type
    detachment.
 2. Vaginal Tear - Circular / Vertical, colporrhaxis.
 3. Extended Episiotomy — upwards towards
    posterior fornix, downwards involving anus
    and rectum.
 4. Vulval Hematoma .
 5. Perineal lacerations.
 6. Para Urethral tear , clitoral tear.
 7. Uterine Rupture – complete / incomplete
 8. Broad Ligament hematoma
Cervical Tear Repair
 Recognition - unilateral / bilateral
 Stitching under good light,
VulvaL hematoma
Spntaneous rupture uterus
Treatment Protocol for Rupture
Treatment protocol of Uterine rupture
3rd T -           TISSUE FACTOR
 Retained Placenta
    1. With Active Bleeding---MRP
       - Partially Separated.-----MRP
       - Retained Cotyledons.----Uterine Exploration and E&C
      - Retained piece of Membranes.—Uterine Exploration and
         E&C
    2. With No Bleeding.
       - Active Retention ( Hour Glass Contraction)-G.A., Placental
          Delivery.
       - Placenta Accreta
       - Placenta Inccreta
        -Placenta Perccreta.
      Acute inversion of Uterus--Protocol
Retention of Placenta
Retention of                            Retention of
Detached Placenta                        Adherent Placenta




Uterine     Hour glass contraction   Simple Adhesion      Morbid Adhesion
Inertia     ( constriction ring )                      Placenta Accreta
                                                       Placenta Inccreta
                                                       Placenta Perccreta
Morbid Adherent Placenta
 Placenta Accreta Rare occurs in 1: 500 to
  1: 700 deliveries. Placenta adhers to uterine
  wall because the Decidua and the Nitabuch
  layer , the physiological cleavage in decidua is
  lacking or incomplete.
 Inccreta placenta penetrates the uterine
  myometrium to variable3 depth.
 Perccreta Very rare occurring in1 : 6,000 to
  1: 40,000 deliveries. Placenta perforates the
  entire thickness of uterine wall and serosa
  even., -- entire placenta or part of it maybe
  morbidly adherent.
A hysterectomy specimen of placenta Percreta
Placenta Perccreta
( A case of Placenta Previa & rupture Uterus)
Rx of Morbid Adherent Placenta
  Once the diagnosis is made , counseling the woman
   and her relatives regarding possible need of Hystere
   ctomy will avoid psychological and medicolegal
   problems.
  Medical Management Umbilical cord is cut close
   to placenta and left in situ. A course of 6 doses of
   Methotrexate is given orally or parenterly in a dose
   of 50 mg Methotrexate and 6mg Folinic acid on
   alternate days.
  Follow up with USG and Beta HCG estimation
   weekly indicates the need for further courses of the
   medicine.
  Woman can have normal Delivery in future.
Inversion Of Uterus
 “Turning inside out of the uterus”
 Acute Inversion is extremely rare ---In India its incidence
  is reported ----1: 23,0000 deliveries.
 Management
    1. Resuscitation measures must be promptly instituted ,
  Blood transfusion , I.V.fluids and sedation given.
   2.Immediate manual reposition in labour room under
  sedation ( 2- 4.gm MgSo4 in 10ml iv or Terbutalin o.25
  in5ml saline iv.)without administration of utero -tonics .
   3.O “ Sullivan ‘s technique of intra vaginal hydrostatic
  pressure.
   4. Surgical Technique---usually required in chronic
  case3s.
Faulty Technique of placental delivery leading to
            Inversion Uterus(a,b,,c)
Acute complete Inversion of Uterus with Attached Cord
Diagramatic Staging Of Inversion OF Uterus
Abdominal view of Inversion Of Uterus
Manual Reposition Of Inverted Uterus
Treatment Protocol for Inversion Uterus
Treatment Protocol for Inversion Uterus
Saving life of the one giving birth
to a new life…
THANKS

Surgical management of pph at tertiary center

  • 1.
    SURGICAL MANAGEMENT OFPPH AT TERTIARY CENTER PROF. (Dr ) M.C. Bansal MBBS, MS , FICOG , MICOG .
  • 2.
    Pregnancy the most dangerousjourney of mankind…
  • 3.
    Definition WHO defines PPHas blood loss of more than 500 ml following vaginal delivery or more than 1000 ml after caesarean section. In Asian women even loss of 300 ml can have sinister effect due to smaller built, lesser blood volume, lower Hb & poor nutrition. However, various authors suggest that PPH should be diagnosed with any amount of blood loss that threatens the hemodynamic stability of the woman.
  • 5.
    Causes of MaternalDeath Haemorrhage 24.8% Infection 14.9% Haemorrhage Indirect is the biggest causes and fastest 19.8% killer Eclampsia 12.9% Other direct causes Obstructed labour Unsafe 7.9% 6.9% abortion 12.9%
  • 6.
    Postpartum Hemorrhage  PPHis a serious, Life-threatening obstetric problem.  One of the leading causes of maternal morbidity and mortality.  In developing countries mainly due to three delays: - 1. Delay in seeking care. 2. Delay in reaching care. 3. Delay in receiving care.
  • 7.
    Maternal Mortality followingPPH WHO estimated 5,29,000 maternal death / year in world 1,36,000 maternal death/ year in India i.e. 30% 29.6% maternal deaths due of PPH. In India, about 15.15% - 25.8% mothers die due to PPH.
  • 8.
    Additional burden ofPPH • > 50% of births in India take place at home without the help of trained/ qualified Birth attendant . • 83% of rural deliveries occur at home • 80% of women are anaemic
  • 9.
    Incidence of PPH •11% women with live birth i.e. 14 million women / year • 3.9% in vaginal deliveries • 6.4% in Cesarean section . • Higher with high risk factor • 10% overall. • Mismanagement of III stage results in higher incidence of PPH
  • 10.
    The Four TsMnemonic – Causes of PPH Four Ts Causes Incidence (%) 1st Tone Atonic uterus 70 2nd Lacerations, hematomas, 20 Trauma inversion, rupture 3rd Tissue Retained tissue, 10 Invasive placenta 4th Coagulopathies 1 Thrombin Am Fam Physician 2007;75:875-82.
  • 11.
    MMR & MORBIDITYDUE TO OBSTETRICAL HAEMORRHAGE
  • 12.
    ‘Prevention is easier andbetter than cure’
  • 13.
    Prevention of PPH??? It can be achieved by Active management of 3 rd stage of labour
  • 14.
    Active Management –Why? 30 20 Percent 10 0 Transfusion Prolonged 3rd Therapeutic Low Retained Stage Uterotonic Hemoglobin Placenta Active Management Physiological Management McMormick, Sanghvi, Kinzie, McIntosh, IJGO2003 1. Reduces length of time of 3rd stage 2. Reduces amount of blood loss 3. Reduces need for blood transfusions
  • 15.
    Prevention Universalization of Spancer’s Modification of Brand- Andrew’s technique of placental delivery e.g. prophylactic injection of uterotonics at the time of delivery of anterior shoulder followed by traction and counter traction maneuver for placental delivery.  Prompt recognition and aggressive management according to the cause of PPH.
  • 16.
    Prevention (contd) Shout for help   1. Blood bank , relative, donors, blood and blood components.  2. Anesthetist  3. Prepare Operation Theater.  4. Immediate Communication with nearest/ dependent Tertiary center,  5. Quick transportation to Tertiary Centre (summon obstetrical flying squad /108 Ambulance)
  • 17.
    Recognition Referral Responsiveness
  • 18.
    “No matter wherea woman delivers, giving birth should be a moment of joy, not a sentence to death”
  • 19.
    “While managing PPH Timelapsed should not be counted in a minute- --one has not lost one minute ,but 60 seconds” Ian Donald
  • 20.
    “No amount ofBlood from any blood bank is safer and better than Her own blood.” Hence be prompt in saving each second and every drop of blood of bleeding woman in her 3rd stage of labour,
  • 21.
  • 22.
  • 23.
    Treatment Protocol OfPrimary Atonic PPH (1st T) Management Management of of Shock Uterine atonicity Replacement of blood * Conservative medical or its component management * Surgical management - Conservative surgery - Radical surgery
  • 24.
    Stepwise Management ofAtonic PPH Step I - Bleeding continues - 15 methyl PGF2 250g every 15-30 mint. Step II - a) Bimanual compression b) Aortic compression Step III - Transvaginal options - Uterine packing - Tamponade Step IV - Compression sutures B.Lynch, Hayman, Cho Square Step V -Other surgical measures - stepwise uterine devascularisation Step VI - Hysterectomy
  • 25.
    Conservative Surgical Management Modeof Actions:  Controls PPH  Preserves reproductive functions  Avoids hysterectomy and related complications and consequences
  • 26.
  • 27.
    Conservative Surgical Management Options In Atonic PPH  Manual removal of placenta  Exploration of uterus  Uterine Packing by Roller gauze, Condom, Foley Catheter, Sengstaken Blackmore tube, Surgical Glove distention,Balloon tamponade  Step-wise devascularization of uterus  B-Lynch suture, Hayman suture, Gun Sheela, Cho multiple square suture  Internal iliac (hypogastric) artery ligation  Postpartum Arterial embolisation
  • 28.
  • 29.
  • 30.
    UTERINE PACCKING PLASTCBAG INFUSED WITH SALINE
  • 31.
    Balloon Tamponade  Two-waycatheter - temporary control of PPH  Feasible in a scenario of atonic PPH following a vaginal delivery, unresponsive to medical management & before interventional radiological procedures or surgical interventions  Simple, cheap, easy to use & effective measure
  • 32.
    Procedure  Balloon portionis placed directly into uterus [entire balloon (500ml capacity) has to be inserted past the cervical canal & internal os].  Gentle traction on balloon shaft ensures proper contact between balloon & tissue surface & enhances tamponade effect  Success is judged by a declining loss of blood from cervix & that seen through drainage port  Mean time for leaving the tamponade balloon - 8 to 48 hours  Gradual deflation of the balloon is advised to reduce the potential risk of further bleeding
  • 33.
    Step-Wise Devascularisation OfThe Uterus  1st reported from Egypt  Effective in controlling PPH in 80% of cases  Unilateral uterine artery ligation  Bilateral uterine artery ligation at the upper part of the lower uterine segment  Low uterine vessels ligation after mobilization of the bladder  Unilateral ovarian vessel ligation  Bilateral ovarian vessel ligation
  • 35.
    Ovarian Artery Ligation Ovarian artery directly arises from the aorta  Anastomosis with the uterine artery in the region of the uterine aspect of the utero-ovarian ligament
  • 36.
    Uterine Artery Ligation 90% blood supply of uterus in pregnancy is from uterine vessels  Ligation of uterine arteries result into significant reduction in blood flow to the uterus
  • 37.
  • 39.
    Vaginal Route for Uterine Artery Ligation  Indicated in atonic PPH following vaginal delivery
  • 40.
    B-Lynch Suture  Exertscontinuous vertical compression on uterine vascular system  Before proceeding to place the suture into uterus, potential efficacy of B-Lynch suture should be tested for by performing open bimanual compression to see if bleeding stops  The assistant performs compression & maintains it with 2 hands during the placement of the suture by the surgeon  Monocryl suture or Vicryl number 2 should be used
  • 41.
  • 42.
  • 43.
  • 44.
  • 45.
  • 48.
    Cho Multiple Square Compression Sutures  Multiple square sutures are used to cover the whole body of uterus using a straight 10-cm needle  May be useful in placenta previa
  • 49.
    Sketch of pelvicblood vessels
  • 50.
  • 51.
    INTERNAL ILLIAC ARTERY(ANATOMY) SURGICAL DISSECTION
  • 52.
    Internal Iliac ArteryLigation Conditions indicating ligation –  Atonic uterus refractory to other measures  Abruptio placentae with uterine atony  Abdominal pregnancy with pelvic implantation of the placenta & placenta accreta
  • 53.
    Internal Iliac ArteryLigation T Therapeutic indications  Before or after hysterectomy for PPH  Continuous bleeding from the broad ligament base; profuse bleeding from pelvic side-wall or vaginal angle  Diffuse bleeding without , clearly identifiable vascular bed  Ruptured uterus in which uterine artery may be torn at its origin from internal iliac artery  Where extensive lacerations of cervix have occurred following difficult instrumental delivery
  • 54.
  • 56.
    Uterine Artery Embolization Highly feasible, safe & beneficial procedure, possibly precluding further laparotomy & hysterectomy  If successful, not only saves the patient’s life, but also preserves the functions of uterus ,tubes and ovaries.  Should be the procedure of choice for PPH prior to surgical intervention
  • 57.
  • 58.
    BLOCKED uterine ARTERY after EMBOLIZ ATION
  • 59.
    Hysterectomy  Best immediateoption  When uterine atony is unresponsive to uterotonics  Where facilities for embolization are not available  Obstetrician not well versed with technical aspects of conservative surgical procedures or iliac artery ligation  Indications  Uterine rupture secondary to obstructed labor  Previous Caesarean section  If rupture is extensive & hemorrhage cannot be contained by suture of ruptured area
  • 60.
    PPH in CASEof ABRUPTIO PLACENTA ( Covouliare Uterus)
  • 61.
    2nd T SurgicalTreatment of Traumatic PPH Causes  1.CervicalTear-Lateral, annular, bucket handle type detachment. 2. Vaginal Tear - Circular / Vertical, colporrhaxis. 3. Extended Episiotomy — upwards towards posterior fornix, downwards involving anus and rectum. 4. Vulval Hematoma . 5. Perineal lacerations. 6. Para Urethral tear , clitoral tear. 7. Uterine Rupture – complete / incomplete 8. Broad Ligament hematoma
  • 63.
    Cervical Tear Repair Recognition - unilateral / bilateral  Stitching under good light,
  • 64.
  • 65.
  • 66.
  • 67.
    Treatment protocol ofUterine rupture
  • 68.
    3rd T - TISSUE FACTOR  Retained Placenta 1. With Active Bleeding---MRP - Partially Separated.-----MRP - Retained Cotyledons.----Uterine Exploration and E&C - Retained piece of Membranes.—Uterine Exploration and E&C 2. With No Bleeding. - Active Retention ( Hour Glass Contraction)-G.A., Placental Delivery. - Placenta Accreta - Placenta Inccreta -Placenta Perccreta.  Acute inversion of Uterus--Protocol
  • 69.
    Retention of Placenta Retentionof Retention of Detached Placenta Adherent Placenta Uterine Hour glass contraction Simple Adhesion Morbid Adhesion Inertia ( constriction ring ) Placenta Accreta Placenta Inccreta Placenta Perccreta
  • 70.
    Morbid Adherent Placenta Placenta Accreta Rare occurs in 1: 500 to 1: 700 deliveries. Placenta adhers to uterine wall because the Decidua and the Nitabuch layer , the physiological cleavage in decidua is lacking or incomplete.  Inccreta placenta penetrates the uterine myometrium to variable3 depth.  Perccreta Very rare occurring in1 : 6,000 to 1: 40,000 deliveries. Placenta perforates the entire thickness of uterine wall and serosa even., -- entire placenta or part of it maybe morbidly adherent.
  • 74.
    A hysterectomy specimenof placenta Percreta
  • 75.
    Placenta Perccreta ( Acase of Placenta Previa & rupture Uterus)
  • 76.
    Rx of MorbidAdherent Placenta  Once the diagnosis is made , counseling the woman and her relatives regarding possible need of Hystere ctomy will avoid psychological and medicolegal problems.  Medical Management Umbilical cord is cut close to placenta and left in situ. A course of 6 doses of Methotrexate is given orally or parenterly in a dose of 50 mg Methotrexate and 6mg Folinic acid on alternate days.  Follow up with USG and Beta HCG estimation weekly indicates the need for further courses of the medicine.  Woman can have normal Delivery in future.
  • 77.
    Inversion Of Uterus “Turning inside out of the uterus”  Acute Inversion is extremely rare ---In India its incidence is reported ----1: 23,0000 deliveries.  Management 1. Resuscitation measures must be promptly instituted , Blood transfusion , I.V.fluids and sedation given. 2.Immediate manual reposition in labour room under sedation ( 2- 4.gm MgSo4 in 10ml iv or Terbutalin o.25 in5ml saline iv.)without administration of utero -tonics . 3.O “ Sullivan ‘s technique of intra vaginal hydrostatic pressure. 4. Surgical Technique---usually required in chronic case3s.
  • 78.
    Faulty Technique ofplacental delivery leading to Inversion Uterus(a,b,,c)
  • 79.
    Acute complete Inversionof Uterus with Attached Cord
  • 80.
    Diagramatic Staging OfInversion OF Uterus
  • 81.
    Abdominal view ofInversion Of Uterus
  • 82.
    Manual Reposition OfInverted Uterus
  • 83.
    Treatment Protocol forInversion Uterus
  • 84.
    Treatment Protocol forInversion Uterus
  • 85.
    Saving life ofthe one giving birth to a new life…
  • 86.

Editor's Notes