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Similar to Varicose Veins lecture for medical students
Varicose Veins lecture for medical students
- 1.
- 2. Venous Anatomy ofLower Limbs Superficial venous system Deep venous system Perforator veins
- 3. Venous valves Thevenous valves are abundant in the distal lower extremity and number of valves decreases proximally, with no valves in superior and inferior vena cava Delicate structures Prevent reverse flow in the veins Ensure that the blood is pumped from the superficial to the deep system and back towards the heart when the patient is walking
- 4. Perforator veins Connectsuperficial to deep veins at various levels. Travel from superficial fascia through an opening in the deep fascia before entering the deep veins. The direction of blood flow - from superficial to deep veins. Guarded by valves so that the flow is unidirectional, i.e. Towards deep veins. Reversal of flow occurs due to incompetence of perforators which will lead to varicose veins
- 5. Ankle perforators Lower leg – Cocket perforators Boyd’s Dodd perforators Hunterian perforators
- 6. Varicose Veins Permanentlydilated , elongated veins with Tortuous veins causing pathological ( Reverse flow) circulation. Risk factors Female sex Prolonged standing Raised intra abdominal pressure Increased progesterone High heels
- 7. The etiology ofvaricose veins can be subdivided into three categories: primary, secondary, and congenital (see the image below).
- 8. Classification Of VaricoseVeins Anatomical Long Saphenous System Short Saphenous System Perforator Incompetence Size Of Varices Thread Veins Reticular Veins 1- 4mm Varicosities >4mm CEAP Classification Clinical Etiological Anatomical Pathophysiological
- 9. Thread veins, alsocalled spider veins, are tiny prominent veins just below the skin surface. The best treatment for thread veins is microsclerotherapy
- 11. Valve incompetence /Ch. Venous hypertension Defective microcirculati on RBC diffusion/ lysis Hemosiderin deposition Dermatatis/ capillary damage Chronic Venous ulceration
- 12. Clinical Features Draggingpain, postural discomfort Heaviness in the legs Night time cramps Oedema, itching Discolouration Ulceration
- 13. Cause Of PainIn Varicose Veins Chronic venous hypertension Anoxia Hyperviscosity or red cells Platelet aggregation Capillary functional disorder Altered cutneous microcirculation
- 14. Complications Hemorrhage Pigmentation/eczema Periostitis Venous ulcer Lipodermatosclerosis Talipes equinovsrus DVT Recurrent thrombophlebitis
- 15. Clinical Signs • Saphenofemoralincompetence Brodie-trendelenberg’s test I • Perforator incompetence Brodie-trendelenberg’s test II • DVT Perthe’s test / modified perthe’s • Perforator incompetence Tourniquet’s test • Valvular incompetence Schwartz test • Perforator site localisation Fegan test • Blow outs = perforators Pratt’s test
- 16. Other Examination Abdomenexamination Ulcer Lymphnodal examination
- 17. Investigation In VaricoseVeins Localise the anatomical location of the disease Nature of the lesion Rule out DVT
- 18. Contd… Venous Doppler DUPLEX scan Doppler combined with B mode Ultrasound Functional and anatomical information DVT well made out. Uniphasic signal – normal Biphasic signal – reversal flow
- 19. Contd… Venography Ascending venography • Dorsalvenous arch – canulated • Tourniquet at malleoli • Dye injected • X-rays taken • DVT/perforator status Descending venography • Ascending venogram not possible • Contrast through femoral vein • Valvular incompetence
- 21. Conservative management Elasticcrepe bandage – stockings 30-40mm Hg Elevation of limbs Above the level of heart Graded compression stockings
- 22. Contd.. Unna boot Nonelastic compression Zinc oxide, calamine, and glycerine Dressing changed once in a week Infection should not be there Compression methods Reduce ambulatory venous pressure Trans capillary leakage Improve cutaneous micro circulation
- 23. Medications Calcium dobesilate Improves lymph flow, reduce edema Diosmin Protects venous valves / anti inflammatory Not proven much beneficial
- 24. Sclerotherapy Complete sclerosisof the venous wall Indications Uncomplicated perforator incompetence Smaller varices Recurrent varices Isolated varices Aged/patients not fit for surgery.
- 25. Contd… Sclerosants usedare Sodium tetradecyl sulphate Sodium morrhuate Ethanolamine oleate Polidocanol Mechanism of action Aseptic inflammation Perivenous fibrosis Endothelial damage Obliteration by intimal approximation
- 26. Technique 23 gauge needle into vein and emptied 0.5 -1 ml of sclerosant Immediate compressio n bandage Proper endothelial apposition May have to be repeated after 2-4 weeks later
- 27. Contd… • Saphenofemoral incompetence •DVT • Peripheral arterial disease • Hypersensitivity Contraindication • OPD procedure • No anesthesia Advantages • Anaphylaxis/shock • Abscess • Thrombophlebitis • Intravenous hematoma • Temporary ocular disturbances Disadvantages
- 28. Interventional Procedures Relievesymptoms Pain / discomfort Reverse complication Cosmesis
- 29. Surgical management Trendelenberg’sprocedure Juxtafemoral flush ligation of long saphenous vein Flush ligation of tibutaries Superficial circumflex Superficial external pudendal Superficial epigastric Deep external pudendal Unnamed tibutaries
- 30. Contd… Stripping oflong saphenous vein Upto knee joint Myer’s stripper Complications Saphenous nerve injury Hematoma Infection
- 31. Contd… Perforator incompetence Subfascialligation of perforators-- Linton’s method (The Linton procedure involves making a long incision across the calf including the diseased tissue, forming a skin/soft tissue/fascial flap, and ligating the perforator veins under direct visualization.) Percutaneous endovenous RFA of incompetent perforator veins Stab avulsion method subfascial endoscopic perforator surgery(SEPS)
- 32. SEPS Subfascial endoscopicperforator surgery Minimally invasive method
- 33. Endovenous Laser Ablation- EVLA US guidance LSV canulated above knee Guide wire passed beyond SFJ Tip is placed 1cm distal to SF junction Laser fibre inserted upto the catheter Diode laser used for firing
- 34. Contd… Thermal damageof endothelium – occlusion of vein Laser energy acts on blood – in turn heats the vein wall. Complications Pain / ecchymosis Hematoma Skin burns DVT
- 35.
Editor's Notes
- #8 The cause of primary varicose veins is valvular insufficiency of the superficial veins, most commonly at the SFValve, resulting in venous hypertension. Secondary varicose veins are mainly caused by DVT that leads to chronic deep venous obstruction or valvular insufficiency. Long-term clinical sequelae from this have been called the postthrombotic syndrome. This category also includes catheter-associated DVT. Pregnancy-induced and progesterone-induced venous wall and valve weakness worsened by expanded circulating blood volume and enlarged uterus can compress the inferior vena cava and hinder venous return from the lower extremities. Trauma is another possible cause of secondary varicose veins. The congenital category includes any venous malformations. Examples include Klippel-Trenaunay variants and avalvulia.