2
• Most heartdiseases in young children are
congenital. The cause is rarely known.
• The prognosis in many cases is poor, due
to lack of suitable treatment.
• Older children are more likely to have
acquired heart diseases such as
rheumatic fever, endomyorcardial fibrosis,
e.t.c.
3.
3
• The heartmay also be affected in many
systemic disorders, e.g., infections,
malnutrition, anaemia, electrolyte
disturbances.
• The prognosis and treatment of these are
related to the underlying cause. Adequate
treatment can help in many of these
cases.
4.
4
Congestive heart failure
•Congestive heart failure is inability of the heart
to pump an adequate amount of blood to the
systemic circulation at normal filling pressures to
meet the metabolic demands of the body.
• In children CHF most frequently occurs
secondary to structural abnormalities (e.g. septal
defects).
• CHF can also occur because of excessive
demands on a normal heart muscle, such as
sepsis or severe anemia.
5.
5
Pathophysiology
• Heart failureis often separated into 2 categories:
right sided and left sided failure.
• In right sided failure, the right ventricle is
unable to pump blood effectively into the
pulmonary artery, resulting in increased
pressure in the right atrium and systemic venous
circulation.
• Systemic venous hypertension causes hepato-
splenomegaly and occasionally edema.
6.
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• In leftsided failure, the left ventricle is
unable to pump blood into the systemic
circulation, resulting in increased pressure
in the left atrium and pulmonary veins.
• The lungs become congested with blood,
causing elevated pulmonary pressures
and pulmonary edema.
7.
7
• Although eachtype of heart failure
produces different signs and symptoms,
clinically it is unusual to observe solely
right or left sided failure in children.
• Since each side of the heart depends on
adequate function of the other side, failure
of one chamber causes reciprocal change
in the opposite chamber.
8.
8
• If abnormalitiesprecipitating congestive heart
failure are not corrected, the heart muscle
becomes damaged.
• Despite compensatory mechanisms, the heart is
unable to maintain an adequate cardiac output.
• Decreased blood flow to the kidneys continues
to stimulate sodium and water reabsorption,
leading to hypervolemia, increased workload on
the heart, and congestion in the pulmonary and
systemic circulations.
9.
9
Signs of cardiacfailure
• In the diagnosis of cardiac failure in
children, look carefully for the following
features:
• Tachycardia- rapid pulse. In newborns and
infants up to 1 year, the normal pulse rate
is 80 -160/minute, in older children 70-
120/minute (with 10 extra for every degree
of fever over 370
c).
10.
10
• Tachypnoea- rapidrespiration of more than
60/minute in infancy or more than 40/minute in
older children.
• Dyspnoea- shortness of breath with deeper and
more rapid breathing. The respiratory symptoms
are due to pulmonary congestion (backward
failure of the left heart) dyspnoea is usually
present at rest but always more pronounced
after exertion. In infants, feeding difficulties are
often the presenting complaint.
11.
11
• Oedema andother signs of raised venous
pressure. Enlargement of the liver is the most
frequent and important sign. Peripheral oedema
is an early symptom of cardiac failure in children.
• The cause is congestion with blood unable to
enter the right heart (back ward failure of the right
heart).
• The increased venous pressure also may cause
engorgement of the neck veins.
12.
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• Tiredness andfailure to thrive. These are
caused by insufficient blood circulation.
Retarded development often is found in
small children with congenital heart
disease due to insufficient oxygen supply
to the growing tissues.
13.
13
• The signsand symptoms of cardiac failure
can be divided into 3 groups:
Impaired myocardial function
Pulmonary congestion
Systemic venous congestion.
17
Management of CHF
•Examine the patient carefully for signs of cardiac
failure.
• Try to find out the underlying disease or cause
of dysfunction, e.g. congenital or acquired heart
disease, severe anaemia, severe pneumonia or
renal failure, and treat specifically if possible.
• Any child with congestive heart failure should be
referred to hospital whenever possible.
18.
18
• Check weightof child, record pulse and
respiration carefully at 2 hourly intervals,
and indicate the exact time of any drugs
given.
19.
19
The goals oftreatment are to:
1) Improve cardiac function
2) Remove accumulated fluid
3) Decrease cardiac demands
4) Improve tissue oxygenation and
decrease oxygen consumption.
20.
20
To decrease cardiacdemands
• Bed rest and comfort.
• Try to relieve anxiety.
• Sedation if necessary: diazepam 0.1-
0.3mg/kg/day in 2-3 divided doses.
21.
21
To remove accumulatedfluid and sodium
• Treatment consists of diuretics, possible fluid restriction,
and sodium restriction
• Diuretics: give furosemide (lasix) 1mg/kg I.V in acute
cardiac failure, but long term treatment with diuretics is
rather dangerous and should not be given without
electrolyte control. Spironolactone (K+
sparing diuretic).
To improve tissue oxygenation and
decrease oxygen consumption.
Supplemental humidified oxygen.
22.
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To improve cardiacfunction;
• Digitalization is most important:
• Digoxin is given orally or intramuscularly.
In severely sick or vomiting children, the
i.m route is recommended to start with.
• note: if digoxin is given i.m. or i.v., the
digitalizing dose is 75% of the oral dose.
23.
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Severe anaemia
• Givepacked blood cells 10mls/kg.
• Do not give more fluid than 100ml/kg/day
for older children to avoid unnecessary
work for the heart muscle.
• Find out the cause of anaemia (malaria?
Hook worm? Sickle cell disease? Severe
malnutrition?) and treat accordingly.
24.
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Severe pneumonia
• Giveantibiotics parenterally
• Feed by nasogastric tube
• Suction regularly to avoid airway
obstruction.
• Apply tepid sponging in case of fever.
26
Nursing considerations
• Admittedto the hospital where intensive nursing
care is available.
• Positioned for optimum ventilation and oxygen
administered.
• IV access is established.
• Cardiac and respiratory function is monitored
continuously using a cardiac monitor and pulse
oximeter to monitor oxygen saturation.
• Urine output monitored.